Alcohol Treatment

Florida Detox® – Painless Alcohol Detox – Outpatient Alcohol Treatment

With our scientific alcohol treatment, you do not have to be locked up in an alcohol rehab facility. You will spend 2 hours a day in our clinic receiving our proprietary all natural intravenous amino acid therapy. We will stop your alcohol craving. Furthermore you will not go through any painful alcohol withdrawal symptoms.

Scientific Alcohol Treatment – Stops Alcohol Craving 

Alcohol Detox is only the first step of alcohol treatment. Alcohol Detox is worthless if you relapse. Our scientific treatment will optimize your brain chemistry and stop your alcohol craving.   

Todd is a grateful patient who went through 44 alcohol rehab programs before finding Dr Sponaugle. We stopped Todd’s “biochemical” craving for alcohol in just days.  

Florida Detox and Wellness Institute Patient - Tim - Alcohol Addiction Treatment

Todd relapsed through 44 alcohol rehab programs. He binged alcohol for 32 years between age 12 and age 44. Todd had failed “the best” alcohol rehabs in America because rehab doctors failed to diagnose his inherited brain chemistry deficiencies. Todd spent over $900,000 on alcohol treatment. 

Todd presented to Florida Detox®  in February, 2009. He came in a wheelchair, he could not walk. We stopped Todd’s craving for alcohol and pain pills.

We treated over 60 hormonal, nutritional and brain chemical deficiencies in Todd. Prior to our scientific alcohol treatment, Todd had never abstained from drugs or alcohol more than five days in 32 years. He also stopped drinking 42 cups of coffee a day and has reduced his cigarette consumption from sixty a day to six.

Todd is active in AA and frequently brings other alcoholic patients to our clinic for help. Here are a few testimonials from those patients.

We’ll diagnose the underlying cause of your alcoholism, don’t waste years of your life and your finances on non-scientific alcohol treatment.

Jennifer’s Story:
Jennifer is a 54-year old nurse who relapsed to alcohol just four days after leaving an Arizona rehab center where she spent 28 days and $46,000.
Before age 50, Jennifer drank just one glass of wine with dinner, it was a social drink, not medication.  After Jennifer entered menopause, she suddenly needed alcohol to “quiet her anxious brain. Estradiol modulates the function of two important brain chemicals, serotonin and dopamine.Jennifer’s 4 years of drinking alcohol caused severe serotonin and taurine deficiencies and her excessive histamine levels were causing excessive electrical activity throughout her brain.
Jennifer’s brain chemistry analysis  reveals a classic pattern Dr. Rick Sponaugle observes in all of his alcoholic patients.  Dr. Rick Sponaugle’s research in alcoholic patients like Jennifer has proven they develop severe deficiencies of serotonin and taurine, two calming brain chemicals and, excessive levels of histamine, an excitatory brain chemical.

The Arizona rehab center had just incorporated SPECT brain imaging into their addiction treatment.    

Dr. Rick Sponaugle has used SPECT imaging in alcoholics since 2005. Dr. Rick Sponaugle’s staff advised Jennifer that she should bring her SPECT scan to her first appointment at Florida Detox® & Wellness Institute.

Jennifer’s Brain Scan (surface)

The holes seen at the top of Jennifer’s surface scan represent a localized dopamine deficiency which causes sub normal activity in her brain’s prefrontal cortex, the brain region behind her forehead. This finding on Jennifer’s scan matches her inherited personality profile, one that was over shadowed by changes in personality when she entered menopause.

Jennifer’s localized dopamine deficiency is derived from inheriting a gene that produces a super COMT enzyme, an enzyme that metabolizes or breaks down dopamine four times faster than normal.

Patients who inherit a “super COMT enzyme” metabolize Dopamine before it can activate sequential brain cells in the prefrontal cortex. The holes represent “a string of light bulbs” that have failed to activate.

Jennifer’s SPECT scan  is called a deep scan. The white and red oval in the middle of Jennifer’s scan represents her severely overactive deep limbic system. The deep limbic system resides in the midbrain and is about the size of a walnut. It is larger and more active in women than men, this makes women more sensitive and more adept at discerning social cues than men.

Dr. Rick Sponaugle’s research has proven that excessive activity in the deep limbic system is caused by alcohol induced serotonin and taurine deficiency.

Jennifer also has an overactive basal ganglion [dopamine factory] just above and to the right of her deep limbic system. This can be a normal presentation, if abnormal, it can be derived from excessive dopamine activity.

In Jennifer’s case, her dopamine levels were normal. Her basal ganglia was overactive due to excessive histamine activation derived from alcohol induced leaky gut syndrome. Histamine activates dopamine receptors throughout the brain. This causes generalized anxiety and insomnia.

When the deep limbic system is quiet, we are much more relaxed and much happier. When the deep limbic system becomes overactive, we develop symptoms of depression, moodiness and irritability. If you have recently developed an overactive emotional center, you are experiencing feelings of hopelessness and excessive guilt. You no longer enjoy things that you once considered fun and you generally feel more depressed. You have developed a more negative personality and your desire to socialize is less that before.

During Jennifer’s initial consultation, Dr. Rick Sponaugle noticed considerable tension between Jennifer and her husband. Jennifer’s Arizona rehab doctors had suggested to Jennifer’s husband that her relapse just four days after leaving their rehab center was her fault, that she was not serious about her sobriety. Jennifer insisted that she needed the alcohol to calm her anxious brain and lift her depression.

Jennifer did not know why, but, she insisted alcohol made her “feel more normal and that without it, she felt miserable.

Dr. Rick Sponaugle explained to Jennifer’s husband, a hospital administrator from Atlanta, that Jennifer was most likely being truthful. Dr. Rick Sponaugle explained that Jennifer was suffering “dry drunk” syndrome. Dry drunk syndrome is the miserable state alcoholics feel when alcohol induced brain chemistry deficiencies are left uncorrected. This it is the number one cause of early relapse in alcoholics.

At Florida Detox® & Wellness Institute, Dr. Rick Sponaugle’s addiction research has matched SPECT brain imaging with specific brain chemistry profiles, female hormonal deficiencies and alcohol craving patterns. Alcohol craving is truly biochemical in nature – craving for the biochemical effect alcohol has on the brain.

The pertinent brain physiology regarding menopause is that serotonin receptors in the female brain close and are unavailable for serotonin activation when estradiol levels fall below 60-80 pg/dl. Dr. Sponaugle  recently taught this concept to Dr. Daniel Amen and 200 psychiatrists at an Amen brain conference in Washington D.C.

Women with normal serotonin production, but, post menopausal estradiol levels suffer serotonin deficiency symptoms! Post menopausal women get less benefit from a serotonergic medication like Lexapro because their serotonin receptors are closed and cannot receive serotonin molecules.

When Jennifer turned 50, she reached full menopause and her estradiol production ceased. She began suffering classic symptoms of serotonin deficiency depression with the inability to enjoy things that used to be fun, increased moodiness, irritability, negativity, less desire to socialize, feelings of hopelessness, excessive guilt, and she became more easily offended.

Serotonin is a calming brain chemical – serotonin deficiency causes excessive electrical activity in the brain’s emotional center – the deep limbic system. Jennifer’s brain scan below demonstrates a severely overactive emotional center [deep limbic system].

Jennifer began drinking more alcohol for it’s calming GABA effect. Alcohol activates the Xanax receptor – the GABA receptor. Activation of the GABA brain receptors reduces electrical current in the brain. Alcohol produced a calming effect Jennifer badly needed once she reached menopause –  her menopausal estradiol levels fell too low for her serotonin receptors to accept serotonin.

Once Jennifer began using wine as a medication to calm her overactive brain, she developed a myriad of alcohol induced brain chemistry imbalances. Dr. Sponaugle’s research has proven that Alcoholic patients develop a toxic gut – an intestine that suffers excessive overgrowth of Candida yeast and Klebsiella bacteria. Toxins from these foreign invaders shut down Serotonin production in the small intestine causing a deficiency of this important calming brain chemical.

Enzymatic breakdown of Candida Yeast in the alcoholic gut produces excessive production of Beta-alanine, a protein that competes with Taurine for re-absorption in the kidneys. Taurine is a calming brain chemical. Dr. Rick Sponaugle’s research in alcoholic patients has proven that all alcoholics suffer Serotonin and Taurine deficiencies. Dr. Rick Sponaugle has designed an intravenous protocol to quickly correct deficiencies of these and other brain chemicals to prevent  the alcoholic patient from relapsing.

Dr. Rick Sponaugle’s research has proven that deficiency of these two brain chemicals produces excessive electrical activity in the deep limbic system, a brain region that is often referred to as our emotional center because it stores our emotional memories. You will notice that Jennifer’s SPECT scan reveals severe over activity in this specific brain region.

The toxins from pathogenic yeast and bacteria destroy the intestinal lining allowing abnormal “leakage” of undigested food particles, parasites and toxins. This phenomenon is known as LGS or “leaky gut syndrome.” The alcoholic intestinal wall ”leaks” larger than normal proteins from the gut into the bloodstream. Antibodies naturally attack these ”foreign bodies” like they would a bacteria or virus as they have no business floating around the bloodstream.

The constant antibody attack up-regulates the immune system producing excessive levels of inflammatory cytokines and excessive levels of circulating histamine. The classic “red flush” on the alcoholic’s face is derived from excessive histamine levels. Most psychiatrists are unaware that Histamine is an excitatory or “electrifying” brain chemical. Histamine has a similar chemical structure to dopamine and it can activate the brain’s dopamine receptors causing excessive electrical current throughout the brain – this is a common cause of anxiety and insomnia in Americans.

With knowledge of the Gut – Brain connection, especially the classic alcohol induced changes in brain chemistry revealed through Dr. Sponaugle’s research, we can better explain why – the more an alcoholic drinks – the more “over electrified” their brain becomes – the more they must drink – to quiet their brain!!

Jennifer relapsed to alcohol just four days after leaving alcohol rehab because her Arizona rehab doctors failed to correct multiple biochemical issues that made her crave alcohol so badly that she was planning her next drink in rehab. Even with the assistance of a SPECT scan, they failed to properly diagnose multiple biochemical imbalances that must be corrected immediately to stop alcohol craving.

They had learned from my colleague, Daniel Amen of the Amen Clinic, that increasing serotonin activity would most likely calm Jennifer’s overactive deep limbic system, the brain region that was most significant in causing Jennifer to self-medicate with the GABA effect of alcohol.

Placing Jennifer on Lexapro, an SSRI, serotonin enhancing anti-depressant, failed to calm her anxious brain leaving her with alcohol craving because:

Jennifer’s  menopausal estradiol levels were too low for her serotonin receptors to remain open, they were not available for increased serotonin activation from Lexapro.

Alcoholic patients like Jennifer produce no serotonin in their intestinal factories, therfore her brain’s serotonin storage units were empty. Drugs like Lexapro do not assist patients in making one molecule of serotonin, they work with available serotonin in the brain, in Jennifer’s case, there was very little serotonin availability for Lexapro to work with.

In alcoholic patients, Candida mycotoxins and Klebsiella endotoxins block the first step, the gut step, of serotonin production, the gut conversion of tryptophan into 5-Hydroxytryptophan. 5-HTP undergoes conversion to serotonin inside the brain’s serotonin factories. Normally, the brain’s serotonin factories receive a healthy supply of 5-HTP from the gut, however, in the alcoholic gut, tryptophan is converted into a toxic chemical called kinurenic acid.

Jennifer’s rehab doctors failed to treat the localized dopamine deficiency in Jennifer’s pre-frontal cortex. The estradiol dropout Jennifer suffered with menopause exacerbated her inherited dopamine deficiency –  Estradiol enhances dopamine production and prevents the breakdown of dopamine.

Estradiol enhances tyrosine hydroxylase, the enzyme that converts tyrosine into dopamine and estradiol inhibits monoamine oxidase, the enzyme that metabolizes dopamine.

Furthermore, Jennifer, like all alcoholics, suffered severe Vitamin D deficiency and iron deficiency. Both Vitamin D and iron are necessary co-factors for tyrosine conversion  to dopamine.

Alcohol stimulates a temporary dopamine surge from brain storage units, with decreased dopamine activity, Jennifer would have craved the dopamine effect of alcohol more than she would have in her premenopausal life.

Jennifer’s rehab doctors failed to diagnose her severe testosterone deficiency and her alcohol induced hypothyroidism. Testosterone in both sexes and Thyroid T3 hormone are necessary for dopamine receptivity in the dopamine driven pleasure [reward center]. The combination of alcohol induced hypothyroidism and menopausal dropout of testosterone and estradiol caused diminished Dopamine D2 activity and subsequent depression that responds to a temporary dopamine hit from alcohol.

Jennifer’s rehab doctors failed to perform extensive hormonal testing and therefore failed to diagnose her alcohol induced adrenal insufficiency. The alcoholic gut toxins are neurotoxins, they readily travel from the gut to the brain and shut down pituitary function.

The reduced production of pituitary hormones TSH [thyroid stimulating hormone] and ACTH [adrenocorticotropic hormone] cause subsequent deficiencies of thyroid hormone, Cortisol, DHEA and Adrenaline. This produces physical fatigue, Jennifer felt like she had a 20 pound cement block attached to each leg. The “dopamine hit” derived from drinking alcohol gave her fake energy.

At Florida Detox® & Wellness Institute, we give our addicted patients a comprehensive Brain Wellness Program that provides evaluation of 250 biochemicals. We optimize and balance all brain chemistry and hormonal issues immediately, this stops alcohol and drug craving.

As of this writing, Jennifer is doing amazing well. She has chosen to continue follow up for through our anti-aging/wellness program. She has now been alcohol free for 2 years and denies any craving for alcohol! She is back on the tennis court and her husband is a happy man!

Visit our blog to read more patient stories.

Our Safe and Painless Alcohol Detox Protects the Brain

We do not allow our patients to suffer any of the typical alcohol withdrawal symptoms associated with alcohol detox. No tremors, no twitching, no spasms, no mini seizures. These alcohol withdrawal symptoms are forbidden in our detox unit because they are indicative of excessive electricity that causes damage to the brain.

You have lost enough brain cells from chronic alcoholism, you can’t afford to lose more going through a dangerous alcohol detox. The loss of brain cells you have sustained from alcohol abuse already increases your risk of getting earlier Alzheimer’s Disease.

Fortunately, our Brain Wellness program has recently reversed Alzheimer’s Disease and is provided to our alcoholic patients at no additional cost. See our Alzheimer’s page under Wellness programs. We can heal brain damage derived from your alcoholism.

Our painless alcohol detox protects your brain from detox induced loss of brain cells.

Recent PET scan studies performed at the University of Toronto have revealed that the old fashioned “shake and bake” alcohol detox performed at most detox centers destroys more brain cells than continuous alcoholism. The University doctors found that the brain of patients who drank alcohol without once stopping in a ten year period sustained better brain volume than the patients who went through alcohol detox once a year.

During your alcohol detoxification, we begin rejuvenation therapy with my intravenous treatment protocol for healing the brain. We replace vitamins, minerals, amino acids and other nutrients that are commonly deficient in alcoholic patients. This aggressive restoration treatment facilitates a much faster recovery and enhances our ability to prevent relapse to alcohol.

Alcohol patients who simply undergo detox, have so many hormonal and biochemical deficiencies, they feel terrible unless they start drinking again. This is called the “dry drunk” syndrome. They might not be drinking, but they feel miserable.

Our six week alcohol treatment program is not mandatory, it is provided at no extra cost to our alcoholic patients. Most of our patients come back for several outpatient appointments, even if they live in California, because they benefit so much from our advanced anti-aging and wellness treatment. Most of our patients look ten to fifteen years younger after just four weeks of this program.

Below is a list of just a few biochemical causes of alcohol craving:

  • Attention Deficit Disorder
  • Anxiety Disorders
  • Depression
  • Insomnia
  • Nutritional Deficiencies
  • Hormonal Imbalance
  • Hidden Food Allergies
  • Allergies to the wheat or corn in the alcohol itself
  • Hypoglycemia
  • Systemic Yeast and Fungal Overgrowth
  • Mold Toxicity
  • Lyme’s Disease
  • Bartonella
  • Babesia
  • Epstein Barr
  • etc.

If you have already been to a rehab center for alcoholism, you know they did not even look for most of these.

We do not discount the fact that patients use alcohol to anesthetize emotional pain, however, we have never treated an alcoholic patient who did not also have many treatable biochemical issues that also caused alcohol craving. We refer those alcohol patients who are suffering  severe emotional pain to some of the best counselors in America.

Isn’t it time you chose scientific alcohol treatment that stops your alcohol craving?

(888) 775-2770

Jennifer’s immediate relapse to alcohol could have been avoided if  the doctors at her Arizona Rehab Center were more knowledgeable about female hormones, particularly their modulation of electrical function in the female brain.
Dr. Sponaugle stopped Jennifer’s alcohol craving in just five days, he knew within 15 minutes of  her initial consultation that Jennifer’s depression and anxiety with subsequent alcohol abuse began when she experienced menopausal cessation of estrogen production.

Jennifer’s history of alcohol abuse tells the real story.

Alcohol Withdrawal

Many chronic alcoholics become maintenance drinkers, drinking daily, to avoid withdrawal symptoms. Withdrawal symptoms can include anxiety, irritability, nervousness, weakness, insomnia, appetite loss, gastrointestinal distress, elevated blood pressure, tremors, seizures and hallucinations. Unfortunately, repeated unmedicated alcohol withdrawal can produce an aggravation of tremors and depression called “kindling.” Repeated alcohol withdrawal and relapse may be more damaging to the alcoholic’s health, than continued maintenance drinking. Robert Post, at the National Institutes of Health, found kindling can also occur with epilepsy and depression.

Many traditional alcohol detoxification programs prevent only grand mal seizures. These programs often allow the nausea, gastric distress, anxiety, insomnia, nervousness, tremors, rapid pulse and unhealthy blood pressures to continue, during detoxification. PET brain scan studies prove merely preventing grand mal seizures, during alcohol detoxification allows increased nerve membrane potentials and voltage to damage neurons, causing permanent damage. Allowing permanent brain damage to occur during alcohol detoxification is cruel and unnecessary. Why is this allowed? Skilled anesthesia eliminates any tremors or twitching during detoxification.

Dr. Sponaugle is a Board Certified Anesthesiologist and has perfected a technique which prevents these unpleasant and dangerous symptoms. In many alcoholics, the severity of withdrawal symptoms increases after repeated withdrawal episodes. This exacerbation may be attributable to a kindling process. Kindling is a phenomenon in which a weak electrical or chemical stimulus, which initially causes no overt behavioral responses, results in the appearance of behavioral effects, such as seizures, when it is administered repeatedly. Both clinical and experimental evidence support the existence of a kindling mechanism during alcohol withdrawal. Withdrawal symptoms, such as seizures, result from neurochemical imbalances in the brain of alcoholics who suddenly reduce or cease alcohol consumption. These imbalances may be exacerbated after repeated withdrawal experiences. The existence of kindling during withdrawal suggests that even patients experiencing mild withdrawal should be treated aggressively to prevent the increase in severity of subsequent withdrawal episodes. Kindling also may contribute to a patient’s relapse risk and to alcohol-related brain damage and cognitive impairment. Becker, H, (1998), p 25.

While the alcoholic is sedated with alcohol, the body up regulates excitatory neurotransmitters including acetylcholine, epinephrine, and glutamate. At the same time, feedback mechanisms down regulate GABA (gamma-aminobutyric acid), which is the most abundant inhibitory neurotransmitter in the brain. Magnesium is also severely depleted by alcohol. Magnesium is necessary to relax muscles and used in hospital treatment of delirium tremors, which can be fatal, if untreated. During alcohol withdrawal, tremors and seizures may occur, since the excitatory neurotransmitters acetylcholine, glutamate and epinephrine are elevated, while the inhibitory neurotransmitter GABA and serum magnesium levels are decreased. Some of the medications used to reduce severity of tremors are Gabapentin, Topiramate and Clonidine. Gabapentin is an anticonvulsant, closely resembling the inhibitory neurotransmitter, GABA. Gabapentin may be used, when liver damage is present, since it is eliminated, in the urine, and not metabolized by the liver. Topiramate is a newer anticonvulsant which is also non addictive. Clonidine (Catapres) is used to reduce the elevated blood pressure and rapid heartbeat of alcohol withdrawal.

Medications which can reduce craving and increase abstinence include Acamprosate, Naltrexone and Topiramate. Acamprosate, recently approved by the Food and Drug Administration, has been used for over 10 years in Europe to manage increased anxiety from alcohol abstinence and has been proven to increase sobriety rates. Acamprosate appears to reduce pre-synaptic release of glutamate. Acamprosate may be used, when liver damage is present, since it is eliminated, in the urine, and not metabolized by the liver. Naltrexone is an opiate antagonist and has been proven to reduce alcohol craving and relapse rates. Topiramate has also been proven to increase abstinence rates and appears to augment GABA levels.

Alcohol Dependence

Alcoholism, also known as “alcohol dependence,” is a disease that includes four symptoms:

  • Craving: A strong need, or compulsion, to drink.
  • Loss of control: The inability to limit one’s drinking on any given occasion.
  • Physical dependence: Withdrawal symptoms, such as nausea, sweating, shakiness, and anxiety, occur when alcohol use is stopped after a period of heavy drinking.
  • Tolerance: The need to drink greater amounts of alcohol in order to “get high.”

People who are not alcoholics sometimes do not understand why an alcoholic can’t just “use a little willpower” to stop drinking. However, alcoholism has little to do with willpower. Alcoholics are in the grip of a powerful “craving,” or uncontrollable need, for alcohol that overrides their ability.Although some people are able to recover from alcoholism without help, the majority of alcoholics need assistance. With treatment and support, many individuals are able to stop drinking and rebuild their lives.Many people wonder why some individuals can use alcohol without problems but others cannot. One important reason has to do with genetics. Scientists have found that having an alcoholic family member makes it more likely that if you choose to drink you too may develop alcoholism. Source: U.S. Department of Health and Human Services. National Institute on Alcohol Abuse and Alcoholism. (2001, January 1). Alcoholism: Getting the Facts (NIH Publication No. 96-4153)[Brochure]. Washington, DC: U.S. Government Printing Office.


Considerable evidence indicates most alcoholics inherit strong genetic propensities toward alcohol addiction. Schuckit, M.,et. al.,(1972) studied 133 sons of alcoholics, adopted and raised by non-alcoholic parents. The 133 sons of alcoholics, raised by non-alcoholic parents, were three times more likely to be alcoholic, than children of non-alcoholic parents. Abnormal electrical activity has also been found, in young sons of alcoholics. P300 brainwaves are frequently studied, in response to mild stimuli. P300 brain waves have been found to weaker or absent in alcoholics and their non-drinking children. Berman, et. al. (1993) reports P300 wave measurements predicted alcohol and drug use 4 years later, (at age 16) in a group of 36 preadolescent boys.Genetic differences also cause alcohol to affect alcoholics differently. Dopamine is possibly the world’s most addictive substance and makes cocaine and nicotine especially addictive. Dopamine creates pleasurable feelings and increases, during sex or when we “fall in love.” Alcohol temporarily elevates dopamine, but depressed dopamine levels occur during alcohol withdrawal. Blum, K et. al., (1996) researched genetics of the dopamine type 2 (D2) neurotransmitter receptor gene and found differences between alcoholics and non-alcoholics. Of 35 alcoholics studied, 69% had the A1 allele, while only 20 percent of 35 non-alcoholics had the A1 allele. These genetic variations determine the number of D2 neurotransmitter receptors.”I have never encountered a single patient who wants to be addicted. They are addicted at enormous personal and social cost, but something fundamental has changed in their brains so that the drives that normally motivate others are disrupted by the drugs,” said Nora D. Volkow, MD, Director of the National Institute on Drug Abuse. . .Dr. Volkow described her study of 20 hospitalized cocaine abusers who showed markedly decreased reductions in dopamine D2 receptors, compared with 23 controls. The diminution of receptors was even apparent four months after cocaine discontinuation. These findings extended the results of other studies showing dopamine D2 receptor reductions in current cocaine abusers as well as studies showing reductions in D2 receptors in other addictions such as alcoholism, heroin, and methamphetamine. “This study documented for the first time the biochemical changes in the brain of addicted people, and showed that the reductions [in dopamine D2 receptors] are long-lasting,” she said. Addicts have fewer dopamine D2 receptors in several regions of their brains than do controls. . . Upon release, dopamine is almost immediately removed from the synapse by the dopamine transporter, and thus has a very limited opportunity to reach the receptor. Fewer receptors for the same amount of released dopamine means less “received” dopamine and consequently less dopamine signaling. . .Having more dopamine D2 receptors in the brain may confer protective benefit against vulnerability to addiction, Dr. Volkow noted. She and her colleagues have measured the number of dopamine D2 receptors in a group of non addicted subjects, then administered intravenous methylphenidate. The subjects who described their experience as pleasurable had significantly fewer dopamine D2 receptors than those who described it as non pleasurable. Liberating more dopamine triggered an aversive experience because it boosted dopamine levels “over the threshold of what would be considered reinforcing.” The experience was too intense and so unpleasant that these subjects refused to return for a follow-up study, Dr. Volkow reported.”Yasgur, B. The Neurologic roots of Addiction. NeuroPsychiatry Reviews. Vol. 5, No. 5; July 2004PET brain scans reveal chemical differences in the brain between addicts and non addicts.The normal images in the bottom row come from non-addicts; the abnormal images in the top row come from patients with addiction disorders. The PET scans from the cocaine abuser, the alcoholic, and the obese patient with food addiction show reduced levels of dopamine receptors (molecules that transmit pleasure signals in the brain). Low levels of dopamine receptors suggest an under stimulated biochemical “reward system” in the brain. The PET scan from the cigarette smoker with nicotine addiction shows lower levels of monoamine oxidase (MAO), a brain enzyme that regulates dopamine levels. BER researchers are investigating pharmaceutical therapies for curbing or curing addictive behaviors. Some alcoholics have higher levels of the alcohol dehydrogenase type II gene, providing them an increased ability to breakdown alcohol. This is also genetically determined. These individuals appear to comprise approximately 50% of the alcoholic population and can metabolize or breakdown alcohol about 40% faster than other alcoholics and non-alcoholics. Asians tend to have less of the alcohol dehydrogenase enzyme which breaks down alcohol and tend to have intense anaphylactic allergic reactions from drinking alcohol similar to the reactions, which occur when people drink on antabuse. Asians experience a histamine flush, often accompanied by constriction of the bronchial tubes, intense discomfort and headaches. Consequently, the alcoholism rate is very low in the Asian population. In contrast, Native Americans have alcoholism rates, as high as, 80 percent. Native Americans have only had about 200 years of exposure to alcohol in some cases, with little opportunity for alcoholism to be bred out of their gene pool In contrast, some Mediterranean populations have had approximately 7000 years of exposure to alcohol and tend to have very low alcoholism rates whether they are Spanish, French, Italian, Greek, Turkish, Syrian, Lebanese, Israeli, Egyptian, Libyan, or Morroccan. This has more to do with genetics than religion, since religions represented around the Mediterranean include Christianity, Judaism, and Islam. Hopefully, Native Americans will eventually breed alcoholism out of their gene pool, but till then, we need more effective treatments.The first step in alcohol breakdown in the body, is transformation to acetaldehyde, by the alcohol dehydrogenase enzyme. Acetaldehyde is essentially a formaldehyde molecule with a methyl group (CH3) added, and is toxic to any tissue in the body. It obviously has a detrimental effect on the brain. Some alcoholics inherit levels of the alcohol dehydrogenase enzyme, about 40 percent higher then the rest of the world, and break alcohol down to acetaldehyde, faster than other drinkers. Unfortunately their liver cannot break down the increased production of acetaldehyde any faster than other drinkers, and higher levels of acetaldehyde accumulate in their bodies.Part of the excess acetaldehyde accumulation, which is not broken down as rapidly as it is produced, combines with serotonin and dopamine, forming opiate-like molecules called tetrahydroisoquinolones, (THIQ). THIQ attaches to the mu opiate receptor. This individual, unlike other drinkers, becomes more intensely addicted to alcohol because THIQ’s opiate-like effects, reduce anxiety and pain. The resulting anxiety reduction perpetuates alcohol addiction. When this individual becomes sober, not only do they crave the opiate, they are also serotonin or dopamine depleted, which increases depression.Naltrexone is a prescription drug, used to saturate the opiate receptor. In approximately 50 percent of alcoholics, it has been found to reduce or eliminate alcohol cravings, although it is more commonly used to treat opiate addiction.

In view of these facts, it seems appropriate to view the alcoholic with compassion and not contempt. Unfortunately, much of the population does not understand these facts and still treats alcoholics contemptuously.

Alcohol And Depression

Depression is a common cause of alcoholism. Many patients who present to Florida Detox® are simply self-medicating depression with their Vicodin, Percocet, OxyContin, cocaine, or alcohol. These drugs, as well as the alcohol give the patient an immediate dopamine surge for temporary treatment of their underlying depression.  The cause of depression in many of these patients is an inherited biochemical deficiency of one or more happy brain chemicals.  When these patients are accurately diagnosed and treated with appropriate medications, they stop craving pain pills, alcohol, or cocaine.

There are multiple biochemical causes of depression. Interestingly, the incidence or prevalence of depressive disorders in males and females is equal up to age 13.  After females reach puberty and undergo hormonal changes, the incidence of depressive disorders increases 300%.  Hormonal changes can absolutely affect brain chemistry and ultimately lead to addiction problems.

Clinical research at Florida Detox® suggests that many female patients suffer imbalance of both hormonal and brain neurotransmitter chemicals.  Unfortunately, many addiction physicians in this country are unaware that low estrogen levels, specifically low estradiol will decrease serotonin receptivity and activity in the brain. Estrogen dropout either post partum or in mid-life, can create serious depression in female patients with no previous history of depressive disorder. The initial evaluation of new onset depression in a 30-45 year old female should always begin with measurement of estradiol levels.

The primary focus on serotonin deficiency as the main cause of depression has created treatment failure in many depressed and addicted patients. Other happy (excitatory) neurotransmitters are often ignored and some patients become more depressed when treated with medication. The classic depressive disorder patient who presents to Florida Detox® is Susan, a 42-year-old professional female who seeks medical attention for depression from her local physician. Dr. Jones immediately assumes that she would benefit from a serotonin enhancer such as Paxil, Prozac, or Lexapro.  If indeed this patient suffers from low serotonin levels, her depression should respond within 2-4 weeks of treatment with the serotonin enhancer. Typically, prescribed a medication like Paxil (20 mg per day), she returns one month later insisting her depression is worse. Dr. Jones raises the Paxil to 40 mg per day. Frequently these patients are prescribed extremely high doses (60 mg to 80 mg per day) in the physician’s effort to conquer the problem. Unfortunately Dr. Jones disregards the continuous report from the patient that they are not feeling any better and may actually feel more depressed. What is the problem? If serotonin is unilaterally elevated above normal levels with the mediation, the brain will down regulate production of dopamine. This makes the patient with dopamine deficiency even more dopamine deficient. These patients will typically begin to self medicate with dopaminergic drugs like Percocet, Vicodin, or OxyContin to counteract the decreased production. All of these drugs produce increased dopamine activity in the brain’s pleasure center (nucleus accumbens). When these patients are accurately diagnosed with their genetic dopamine/glutamate deficiency and treated with appropriate dopamine/glutamate enhancing medication, they quickly experience cessation of their depression and lose the craving (psychological and biochemical) for drugs and alcohol. Treatment results in better relapse statistics with the application of this scientific approach to addiction and depression. Unilateral elevation of serotonin without dopamine level protection will result in markedly elevated prolactin levels. Prolactin will increase appetite and decrease sex drive.  When dopamine levels are enhanced to normal levels, sex drive will return as will better appetite control.


The symptoms of hypoglycemia in many cases are identical to what Alcoholics Anonymous calls drunk syndrome. Depression, irritability, anxiety, insomnia, impaired concentration, dizziness, and headache are common hypoglycemic symptoms.

Different investigators have found 50% to 96% of alcoholics are hypoglycemic. Poulos, J, et. al. (1979) administered a 6 hour glucose tolerance test to 50 halfway house alcoholics and 50 outpatient alcoholics. All 50 outpatient alcoholics were hypoglycemic and 46 of 50 halfway house alcoholics were hypoglycemic, with one diabetic and 3 pre-diabetic halfway house alcoholics. Whether hypoglycemia is a result of their alcohol consumption or they were treating their hypoglycemia with alcohol is perhaps irrelevant once they become alcohol dependent.

Hale F., et. al., (1981) measured mental performance of 67 patients during a five hour glucose tolerance test. Hypoglycemic patients required significantly (p=0.0002 ) more time to perform serial sevens subtractions than non-hypoglycemic patients.

Hypoglycemia is evident at Alcoholics Anonymous meetings. Commonly, regulars are seen drinking colas and other soft drinks, which contain up to 10 teaspoons of sugar. They can also be seen filling a styrofoam coffee cup one-quarter to half-full of white sugar, then topping it off with regular coffee. This will stimulate insulin and glycogen release to rapidly raise blood sugar.

Bill W., cofounder of Alcoholics Anonymous, was apparently hypoglycemic and became increasingly interested in the relationship between hypoglycemia and alcohol. In 1968, Bill W. pleaded with physicians in a 48 page, 8 1/2″ X 11″ booklet to adopt the hypoglycemia diet and vitamins, especially B3, in the treatment of alcoholism.

The crux of the alcoholic problem is hypoglycemia-whether it occurs in the predisposed person with genetic factors influencing his physical and emotional growth, in the social drinker aggravating his already disturbed carbohydrate function, in the chronic alcoholic in the General Adaptation Syndrome (Hans Sleye, M.D.) stage of resistance or fatigue, or in the recovered alcoholic with his psychic and physical complaints. Even in the normal person, a temporary hypoglycemia develops following a debauch. “Vitamin B-3 Therapy; A Second Communication to A.A.’s Physicians From Bill W.” February, 1968.

“The victim is alternately whipsawed between too much insulin and too much adrenalin. Unconsciously, we alcoholics try to cure these conditions-first by sweets, and then by coffee. The sweets temporarily raise our blood sugar, and we feel better. Coffee also gives us a temporary boost because it lessens the shock of the blood sugar drop. In exactly the wrong way, we are unconsciously trying to treat ourselves for hypoglycemia. If you are on B-3 and Vitamin C already, then add the dietary discipline. If you have hypoglycemia to any extent, the dividends are apt to be very large.”
Hypoglycemia Association, Inc. Bulletin #200 July/Aug/Sept.1996 Box 165, Ashton, MD

Glucose is derived from three sources: food; from synthesis (manufacture) in the body; and from the breakdown of glycogen, a form of glucose, stored in the liver and muscles. Hormones help to maintain a constant concentration of glucose in the blood. This is especially important for the brain because it cannot make or store glucose but depends on glucose supplied by the blood. Even brief periods of low glucose levels (hypoglycemia) can cause brain damage.

Two hormones that are secreted by the pancreas to regulate blood glucose levels are insulin and glucagon. Insulin lowers the glucose concentration in the blood; glucagon raises it. Because prevention of hypoglycemia is vital for the body, several hormones from the adrenal glands and pituitary back up glucagon function. Alcohol consumption interferes with all three glucose sources and with the actions of the regulatory hormones. Chronic heavy drinkers often have insufficient dietary intake of glucose. Without eating, glycogen stores are exhausted in a few hours. In addition, the body’s glucose production is inhibited while alcohol is being metabolized.

The combination of these effects can cause severe hypoglycemia 6 to 36 hours after a binge-drinking episode. Even in well-nourished people, alcohol can disturb blood sugar levels. Acute alcohol consumption, especially in combination with sugar, augments insulin secretion and causes temporary hypoglycemia. In addition, studies in healthy subjects and insulin-dependent diabetics have shown that acute alcohol consumption can impair the hormonal response to hypoglycemia. ALCOHOL ALERT. National Institute on Alcohol Abuse and Alcoholism No. 26 PH 352 October 1994

Alcohol is a very high calorie substance. Fats contain 8 calories per gram, which are released slowly. Proteins and carbohydrates (including white sugar) contain 4 calories per gram. Alcohol contains 7 calories per gram and 20% of the alcohol is absorbed through the stomach lining. For the hypoglycemic with low blood sugar, experiencing uncomfortable symptoms, alcohol is the quickest way to increase blood sugar.

Hypoglycemia is fairly easily identified with the five or six hour glucose tolerance test. Severe hypoglycemia is usually evident within three hours. Hypoglycemia often responds well to chromium, zinc, glutamine, and magnesium supplementation and sometimes to vitamin B3 supplementation. Deficiencies of the stress hormone cortisol can also cause hypoglycemia.

Rogers and Pelton, (1957) found glutamine supplementation reduced anxiety, desire to drink and improved sleep, in a 6 week double blind crossover trial, with 7 men and 3 women. Glutamine produced improvements in 9 out of 10 drinkers who used it. During the crossover phase of the study, only 2 or 3 subjects responded to the lactose placebo.

Glutamine is the most abundant amino acid, in the human body and the body contains approximately 100,000 milligrams of glutamine. Glutamine probably helps the alcoholic in at least three different ways. It can be transformed into blood glucose and blood glucose is the only fuel of the brain, which uses at least 20% of the body’s energy and requires a constant supply of blood glucose. Unlike fats and carbohydrates, when glutamine is not metabolized to supply blood glucose, glutamine increases protein production and release of human growth hormone. Glutamine also can transform to the excitatory neurotransmitter glutamate or the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) depending on what arousal level is needed. Glutamine also speeds repair of the intestinal lining, which alcohol damages.

Food Allergies

Food Allergies Cause Alcoholism

Alcohol addiction can also be perpetuated by hidden food allergies. Food allergies often produce cravings for the foods causing the allergy. William Philpott, MD, PhD who is Board certified in Neurology, Allergy and Psychiatry, explains the body produces endorphins or enkephalins, when stressed by an allergen. The opiate like endorphins, reduce anxiety and perpetuate the addiction.

Joseph Beasley, et. al., (1991) tested 108 alcoholics for allergies in a treatment program in Amityville, New York and found allergies in the following percentages: corn, 75.9 percent; milk, 74.1 percent; peanut 67.6 percent; cocoa, 66.7 percent; whole egg, 60.2 percent; orange, 57.4 percent; tomato, 52.8 percent; shrimp, 48.1 percent; and wheat 46.3 percent. Beasley did not test for rye allergy, but other investigators have found a high percentage of alcoholics are allergic to rye. Many beverage alcohols are fermented from corn, wheat and rye. Distillation can concentrate allergenic cogeners, present in these foods. Hidden allergies to these foods can perpetuate these addictions or trigger violent or unpredictable behavior, which is atypical for the drinker.

These allergies can often be detected with an ELISA/ACT serum immunoglobin test. This test can detect delayed Immunoglobin G reactions to over 100 different foods with one blood sample. Since ELISA/ACT testing is performed on a collected blood sample, instead of the skin, it is not necessary to avoid any intake of a food to be tested for a minimum of four days, while the test is performed.

Complete elimination of a suspected food allergen for a minimum of four days, remains the most accurate method to test for hidden food allergy, and may still be necessary, if preliminary testing indicates an allergic reaction. Food elimination testing requires strict diligence avoiding any processed foods containing the suspected food and thorough reading of all ingredients on labels. Since many of these food allergens are difficult to avoid, in typical American diets, allergy testing helps patients realize the necessity of dietary change.

Food allergies often occur, when alcohol damages the intestinal lining. Increased intestinal permeability (leaky gut syndrome) can occur when the intestinal lining is irritated. Undigested proteins can leak through the damaged intestinal lining, and trigger an immune response, when antigen-antibody complexes form. Inflammation occurs when the immune system attacks invaders. These immune reactions can occur, wherever blood transports the antigens, causing depression, headaches, impaired concentration, attention deficit disorder, arthritis and other disorders, which are difficult to associate with the ingested food.

Florida Detox® and Wellness Institute has found corn, wheat and milk allergies in some of our alcohol patients. One of our alcohol patients with a milk allergy, drank a glass of milk daily, before learning he was allergic to milk. When milk allergies are found, more detailed testing can be performed to determine whether the cow milk allergy is to whey, casein, butter, cottage cheese, various other aged cheeses and whether the patient is allergic to goat milk and cheese. Although not technically an allergy, mold neurotoxins can cause extreme anxiety and panic disorders, which some alcohol patients self treate with alcohol. Extensive testing of inflammatory cytokines,anti-inflammatory hormones and inflammation markers, including TNFalpha, alpha MSH, VIP, MMP 9 and activated complement can indicate possible problems with mold, Lymes, Babesia, Bartonella, Mycoplasma or viruses. One female Florida Detox® and Wellness Institute alcohol patient had to move to a new house, after finding a sheet of mold, hidden behind a wall, in her home.

Alcoholics Anonymous

Many alcoholics are unable to successfully “work” the 12 Steps of Alcoholics Anonymous, until nutritional deficiencies and physiological damage, caused by alcohol, are corrected. Nutritional supplementation and medical treatment can reduce or eliminate alcohol cravings, hypoglycemia, essential fatty acid deficiencies, depression, mood swings, anxiety, insomnia, tremors, memory loss, fatty liver, gastritis, gastro-esophageal reflux, mal-absorption, increased intestinal permeability, food allergies, hormonal imbalances, and other problems which prevent problem drinkers from controlling their drinking. Alcoholics Anonymous and 12 Step models continue to dominate most alcohol treatment programs. Their dominance conveys an unwritten message to the public, that 12 Step alcohol treatment programs are the most effective alcohol treatment programs. Alcoholics Anonymous literature has been translated into many languages and many non-smoking meetings are available now. The Big Book of Alcoholics Anonymous was published in 1939 and Alcoholics Anonymous remains essentially unchanged since then.

Unfortunately, Alcoholics Anonymous has ignored medical, scientific and nutritional advances, which are increasingly accelerating. Newer nutritionally or biologically based alcohol treatment programs are producing one year alcohol sobriety rates, as high as 70 to 80 percent. The effectiveness of nutritional alcohol treatment was proven in the early 1950’s and documented by Guenther, R. (1983) who conducted a 6 month study at a Veterans Administration Medical Center, in Waco, Texas, with 108 alcoholic inpatients.

All patients participated in alcohol detoxification and education, AA meetings, group counseling and outpatient follow-up, after a 28 day program. The nutrition experimental group participated in the same program with the same staff, and received nutrition education, nutritional supplements and a more nutritious diet. Dietary modifications included wheat germ and bran with each meal, whole-grain bread, decaffeinated beverages, and sugar substitutes. Unsweetened fruit was used for dessert, instead of cake, pie, pudding or sweetened fruit. Snacks allowed were nuts, cheese, whole-grain bread and peanut butter. Six months after discharge from the hospital, 81 percent of the nutritional experimental group were not drinking, compared to 38 percent of the control group.

The statistical probability of a difference in sobriety rates this large in a group this size was calculated to be two chances in 1000. (p=0.002) (For readers unfamiliar with statistics, p=0.002 means the probability of a difference occurring by chance is 2 in 1000. p=0.05 means 5 chances in 100, or a 5 percent probability of the difference occurring by chance. This probability notation style will be used again later in this article.)If you spend enough time in Alcoholics Anonymous groups, you will probably hear that only about one in four Alcoholics Anonymous participants are sober a year later. Brown, R., et. al. (1990) cited Harrison and Hoffman (1988) who reported 73 percent of alcoholics who attended Alcoholics Anonymous weekly, for at least 6 months were sober, but 30 to 50 percent of alcoholics who initially attended Alcoholics Anonymous dropped out. Brown, R., et. al. (1990) cited Hunt, W. et. al. (1971) who found an average one year alcohol sobriety rate of 33 percent; Bill, C. (1965) who found 34.6 percent of AA members were abstinent, at the end of one year and Milkman and Sunderwirth (1987) who found, “approximately 75 % of all those who attempt abstinence from alcohol reverse their habits between three and six months after beginning a program for recovery.” Some Alcoholics Anonymous groups appear interested in biochemical treatment possibilities. The West Baltimore Alcoholics Anonymous group has linked the following passage, by Abram Hoffer, MD, PhD, to their website. The term vitamin B-3 was reintroduced by my friend Bill W., co-founder of Alcoholics Anonymous, (Bill Wilson). We met in New York in 1960. Humphry Osmond and I introduced him to the concept of mega vitamin therapy. We described the results we had seen with our schizophrenic patients, some of whom were also alcoholic. We also told him about its many other properties. It was therapeutic for arthritis, for some cases of senility and it lowered cholesterol levels. Bill was very curious about it and began to take niacin, 3 g daily. Within a few weeks fatigue and depression which had plagued him for years were gone. He gave it to 30 of his close friends in AA and persuaded them to try it. Within 6 months he was convinced that it would be very helpful to alcoholics. Of the thirty, 10 were free of anxiety, tension and depression in one month. Another 10 were well in two months. He decided that the chemical or medical terms for this vitamin were not appropriate. He wanted to persuade members of AA, especially the doctors in AA, that this would be a useful addition to treatment and he needed a term that could be more readily popularized. He asked me the names that had been used. I told him it was originally known as vitamin B-3.

This was the term Bill wanted. In his first report to physicians in AA he called it “The Vitamin B-3 Therapy.” Thousands of copies of this extraordinary pamphlet were distributed. Eventually the name came back and today even the most conservative medical journals are using the term vitamin B-3. Bill became unpopular with the members of the board of AA International. The medical members, who had been appointed by Bill, felt that he had no business messing about with treatment using vitamins. They also “knew” vitamin B-3 could not be therapeutic as Bill had found it to be. For this reason Bill provided information to the medical members of AA outside of the National Board, distributing three of his amazing pamphlets. They are now not readily available. Niacin, in large megadoses, for prolonged periods can produce liver toxicity. Vitamin B-3, is available as Niacinamide, Inositol Hexaniacinate or Nicotinamide Adenine Dinucleotide Hydrogen, (NADH) which are safer for the liver. Florida Detox does not recommend megadose Vitamin B-3 supplementation for alcoholism, without liver enzyme testing.Despite resistance to change by Alcoholics Anonymous, participation in Alcoholics Anonymous offers considerable advantages to the problem drinker. Alcoholics Anonymous offers alcoholics unparalleled emotional support. If the alcoholic diligently cultivates relationships, in Alcoholics Anonymous groups, it is possible to become acquainted with caring people who work or sleep on a variety of schedules. Sufficient relationships could potentially provide 24 hour per day, seven day per week access to fellowship or emotional support during a crisis. Alcoholics Anonymous is widespread geographically, providing fellowship and support to the problem drinker, during travel.Fellow Alcoholics can often sense when someone is in danger of relapse, earlier than non-alcoholics, or even professional addiction counselors, who were never chemically dependent. Alcoholics tend to have selective memories of their drinking experiences, forgetting the worst disasters, losses, chaos and devastation, while remembering earlier, pleasant alcohol experiences.

Listening to experiences of other problem drinkers reminds them of the devastation and destruction alcohol caused them.The fifth step of Alcoholics Anonymous involves disclosing or sharing your worst mistakes, shortcomings and failures with another person. Sharing these shameful secrets with a non-judgmental person reduces the ability of shame and compulsions to perpetuate undesirable behaviors.

The 12th step of Alcoholics Anonymous may be the most powerful and involves sharing your strength, hope and recovery with other sufferers. This step is a paraphrase of the biblical law of sowing and reaping. As the alcoholic sows recovery into another sufferer’s life, recovery flows into the alcoholics life. An Alcoholics Anonymous sponsor is a mentor who “sponsors” members with less sobriety, by sharing the perspectives and experiences they gained while recovering. Statistically, these sponsors, have the lowest relapse rates, in Alcoholics Anonymous. Sometimes sponsors have been on the verge of a relapse, when they received a call for help, from someone. Alcoholics Anonymous has many catchy slogans, including, “You can’t keep it (recovery), unless you give it away.” When sponsors realize they can transform the worst experience of their life into something powerful, to help another sufferer, their life gains new meaning and purpose. At Florida Detox, many of our recovered patients write testimonials or volunteer to talk with prospective patients, in effect, “working” the 12 th step.

Nutrition Depletion

Nutrient Depletion

Alcoholism leads to Nutrient Depletion. Deficiencies of vitamin B1, B2, B3, B5, B6, B12, folic acid, and biotin have all been found to cause depression, and alcohol depletes all of these vitamins. Alpert, J. and Fava, M. (1997) found 15 to 38 percent of depressed patients were deficient in folic acid. Alcohol depletes all of the B vitamins, Vitamin A, Vitamin C, Vitamin D, and Vitamin E, calcium, magnesium, zinc, potassium and selenium.

Essential Fatty Acid Metabolism

Alcohol increases depression and inflammation by altering essential fatty acid and prostaglandin levels. Alcohol depletes magnesium, zinc, and vitamin B6, interfering with the 6 delta desaturase enzyme. 6 delta desaturase is necessary to convert Omega-3 vegetable oils to eicosapentaenoic acid (EPA) and decosahexanoic acid (DHA) essential fatty acids and Omega-6 vegetable oils to anti-inflamatory prostaglandins (PGE2). Artificially altered hydrogenated vegetable oils, also attach to active binding sites of the 6 delta desaturase enzyme, preventing it from functioning. These hydrogenated fats, previously liquid at room temperatures, are altered by addition of additional hydrogen to the carbon atoms, transforming them from unsaturated to saturated fats. Hydrogenation increases shelf life and spreadability of vegetable oils, margarines and peanut butter. Hydrogenation also straightens bends, in the fat molecule, altering physical configuration. These altered molecules, decrease cell membrane permeability, interfering with neurotransmitter release and reception, nutrient and waste transfer. Hydrogenated fats should be eliminated from the diet.

Omega 3 Fatty Acids

Without 6 delta desaturase enzyme, flax and other Omega 3 vegetable oils cannot be transformed to anti-inflamatory Eicosapentaenoic Acid (EPA). Producing DHA from EPA also requires 6 delta desaturase. Barry Sears, PhD, explains how EPA and DHA reduce heart attacks, stroke, hypertension, arthritis, asthma, emphysema, cancer, infection, Alzheimer’s disorder and depression, in his best selling book, The Omega Rx Zone. DHA, is the most abundant fat in the brain, which is 60 percent fat. In a double blind, placebo controlled study, at Harvard Medical School, Stoll, A., et. al., (1999) compared supplementation with high Omega 3 fish oil to olive oil, in 30 patients with bipolar disorder. In the four month study, patients supplemented with fish oil, performed better on almost every outcome measure, especially time to relapse to depression or mania. At four months, relapse to depression or mania was 47 pecent lower, in subjects using high Omega 3 fish oil. Dosage was 6.2 grams eicosapentaenoic acid (EPA) and 3.4 grams decosahexanoic acid (DHA) daily. Eight subjects in the study were not using any pharmaceutical medication for bipolar disorder and were equally divided into the experimental and control group. The four unmedicated subjects treated with fish oil remained remission free considerably longer than unmedicated placebo subjects. Statisically, the probability of this difference occurring by chance, was 4 in 100. (p=0.04).

Omega 6 Fatty Acids

Omega 6 fatty acid levels are altered in depression and alcoholism. Fortunately, they can be supplemented to decrease depression. Omega 6 oils comprise most of the vegetable oil in the American diet and include soy, sesame, corn, safflower, and sunflower oil. These oils are initially converted to gama-linolenic acid or GLA, by the 6-delta desaturase enzyme. As previously discussed, alcohol interferes with the 6 delta desaturase enzyme. GLA levels are decreased in depressed patients and alcoholics. GLA is a precursor for Dihomo gamma linolenic acid (DGLA) which can be converted to either Arachidonic Acid(AA) or anti inflammatory (PGE1) prostaglandins. AA is a precursor to many inflammatory prostaglandins. When less AA is produced from DGLA, more anti-inflammatory PGE1 prostaglandins can be produced from DGLA. EPA decreases (AA) production by limiting 5 delta desaturase enzyme production. Adams, P, et. al. (1996) found (AA)/EPA ratio increased with severity of depression, in a study of 20 moderately to severely depressed patients. As AA levels decrease, inflammatory prostaglandin production decreases, while anti-inflammatory production increases. One of the inflammatory prostaglandins produced from AA, is PGE2. (Lieb, J., et. al., 1983) measured inflammatory prostaglandin PGE2 in thirty depressed outpatients and found plasma PGE2 levels were elevated in 29 of 30 patients. Anti-inflammatory PGE1 prostaglandins can counteract the inflammatory PGE2 prostaglandins, which were elevated in depression. In summary, increasing EPA levels can reduce depression severity, by suppressing inflammatory prostaglandin production and increasing anti-inflammatory prostaglandin production. Supplementation with fish oil can effectively raise EPA levels and decrease depression.Supplementation with pharmaceutical grade fish oil can effectively bypass genetic or alcohol induced impairment of the 6-delta desaturase enzyme. Since cold water fatty fish oils contain high levels of EPA and DHA, 6-delta desaturase enzyme is not necessary to produce them, from vegetable oils. Newer pharmaceutical grade oils are distilled to remove mercury, polychlorinated biphenyls (PCBs) and other contaminants, which larger fish can bio-concentrate. Burping usually does not occur, since distillation removes “fishy” taste and odors. EPA and DHA are concentrated by distillation, reducing the need to swallow more than a tablespoon per day. The oils are flavored with lemon, lime or orange and actually taste pleasant. Danger of Vitamin D toxicity or overdose is eliminated, if the supplement does not contain cod liver oil. When joint pain, asthma, emphysema or other inflammatory conditions are present, formulas with a higher proportion of EPA to DHA, can be used to counter inflammation.GLA supplementation can also reduce depression and alcohol consumption. Borage, black currant or evening primrose oils can be used to boost GLA levels. David Horrobin, (1985) treated alcoholics in a placebo controlled study with GLA, for one year in Scotland. GLA reduced required tranquilizer doses, withdrawal symptoms, and alcohol craving more than placebo. GLA improved liver function more rapidly and increased one year alcohol abstinence.


Alcoholism alters Tryptophan / Serotonin

Altered tryptophan and serotonin levels occur in alcoholics. Serotonin neurotransmitter deficiency can produce depression. Tryptophan is an essential amino acid, which means the human body cannot produce it, although it is needed, to produce other amino acids. Tryptophan is available by prescription, as L-tryptophan or as an over the counter non-prescription supplement, 5-Hydroxytryptophan. ( 5-HTP ). In the body, tryptophan is transformed to niacin ( Vitamin B3 ) or the neurotransmitter serotonin. If inadequate niacin is present, tryptophan will be converted to niacin, before conversion to serotonin. Conversion of tryptophan to serotonin requires Vitamin B6 ( pyrodoxal 5 phosphate ) and magnesium, as cofactors. Alcohol depletes magnesium and Vitamin B6.

Research by Borg, S, et. al., (1985) indicates alcohol temporarily increases serotonin levels during intoxication, but can decrease serotonin levels for months after the alcoholic becomes sober. Borg measured the serotonin metabolic product, 5-Hydroxyindoleacetic acid ( 5-HIAA ), in cerebrospinal fluid of male alcoholics and controls. 5-HIAA levels increased with blood alcohol levels and gradually decreased with increased abstinence. Subnormal levels occurred in alcoholics abstinent for three months.

Branchey L., et. al., (1985) observed lower plasma tryptophan levels in male patients who had experienced blackouts, compared to patients who had not experienced blackouts. There was no significant difference between proportions of other amino acids which share the tryptophan blood-brain barrier transport carrier. Although increasing serotonin levels can benefit many recovering alcoholics, Bankole, A., et al., (2000) found Odansetron, a 5-HT3 antagonist (which blocks serotonin 3 receptors) reduced drinks per day, approximately 53 percent, in early age onset alcoholics.

Serotonin levels can be increased with l-tryptophan or 5-hydroxytryptophan supplementation, in addition to pharmaceuticals. ( P?nger, W. et. al.,1991) compared 5-hydroxytryptophan ( 5-HTP ) to fluvoxamine ( Luvox ), a selective serotonin reuptake inhibitor antidepressant, in a 6 week double blind study, with 69 depressed participants. Both treatments produced highly significant reductions in Hamilton Rating Scale for Depression, at 2, 4 and 6 weeks. 5-HTP produced greater reductions in insomnia, 61.7 to 55.9 percent, physical symptoms, 47.6 to 37.8, anxiety, 58.2 to 48.3, and depressed mood, 65.7 to 61.8, than fluvoxamine. 5-HTP was better tolerated, with four fluvoxamine patients dropping out of study, compared to one in the 5-HTP group, with 38.9 percent of 5-HTP group reporting side effects, while 54.5 percent of fluvoxamine group reported side effects. Side effects in the 5-HTP group were milder and the sole 5-HTP dropout lasted 5 weeks, while the 4 fluvoxamine dropouts lasted 2 weeks. Dosage was 100 mg 5-HTP three times per day or 50 mg fluvoxamine 3 times per day.

Amen, D and Routh, L. (2003) report brain uptake of 5-HTP is 70 percent, while brain uptake of l-tryptophan is only 3 percent. Additional information concerning depression treatment with amino acids is available, at Amen’s website,


Hypothyroidism can lead to Alcoholism

Hypothyroidism has been estimated to effect up to 10 percent of middle aged Americans and can produce depression. Some hypothyroidism sufferers are unable to convert inactive tetraiodothryronine (T4 ), ( levothyronine,Synthroid) to the active thyroid hormone triiodothryronine (T3), ( liothyronine, Cytomel). Many physicians assume patients who do not respond to low cost generic T4 are not hypothyroid, without attempting a trial of more expensive Cytomel. Frequently, Cytomel will improve hypothyroidism, when T4 medication does not. Cytomel is fast acting, with a short half-life and patients who have not improved with years of T4 medication, often feel better the same day Cytomel is taken. Bipolar disorder which fails to respond to more conventional Lithium or anticonvulsant medications sometimes responds well to T3 medication. Lithium medication can cause hypothyroidism.

The usual laboratory tests for thyroid hormone (TSH, T4, T3) sometimes fail to detect hypothyroidism, especially Hashimoto’s autoimmune thyroiditis, which is becoming increasingly prevalent. More expensive fluorescence activated microsphere assay tests can sometimes detect this condition, where the immune system attacks thyroid tissue.

Jacob Teitelbaum, (2005) reports the American Academy of Clinical Endocrinologists narrowed the normal range for thyroid stimulating hormone (TSH), in November, 2003. A TSH of 3 or higher now indicates hypothyroidism. Since the older criteria considered hypothyroidism to begin with a TSH of 5, instead of 3.0, the number of hypothyroid individuals in the United States, is now estimated to have increased from 13 to 26 million. Teitelbaum states less than 25 percent of these people have been properly diagnosed and treated. Teitelbaum discusses the horrendous human cost of hypothyroidism, in Press Release: The Tragic and Invisible Epidemic of Thyroid Disease.

Depletes Testosterone

Alcoholism Depletes Testosterone

The majority of alcoholics are men and alcohol can deplete testosterone. Many males understand immediately that this could produce depression. Barrett-Connor, E., et. al., (1999) in a study of 856 men, 50 to 89 years old, found Beck Depression Inventory score varied inversely with free testosterone level, independent of age, or physical activity. Bioavailable testosterone was 17 percent lower, for depressed men. Statistically, the probability of a difference that large, was 7 in one thousand. (p=0.007 Alcohol severely depletes zinc and zinc is necessary for testosterone production and prevention of testosterone conversion to dihydrotestosterone, which causes male pattern baldness.

Alcohol is directly toxic to the testes, causing reduced testosterone levels in men. In a study of normal healthy men who received alcohol for 4 weeks, testosterone levels declined after only 5 days and continued to fall throughout the study period (17). Prolonged testosterone deficiency may contribute to a “femininization” of male sexual characteristics, for example breast enlargement (18).

Alcohol Alert National Institute on Alcohol Abuse and Alcoholism No. 26 PH 352 October 1994

Sarkar, D., et. al. (1998) observed, “Alcoholic men show a positive association between the presence of clinically apparent gynecomastia and elevated circulating levels of prolactin. These patients also show an elevation of plasma levels of estrogen, which is believed to be due to peripheral conversion of weak adrenal androgens to estrogen.”

Testosterone has received undeserved bad press in the United States. Gynecomastia or production of female breast tissue in males, is often mentioned as a danger of testosterone supplementation. While almost nonexistent in weight rooms where people are alleged to be abusing steroids, gynecomastia is readily observable in older chronic alcoholics.

Increased testosterone levels appear to prevent benign prostate hypertrophy (BPH). BPH is a non-cancerous prostate gland enlargement, which interferes with urine flow, in older men. When the prostate gland enlarges, it constricts the urethra. Impaired sleep occurs, when men need to urinate frequently during the night. BPH is associated with elevated estrogen and decreased testosterone levels. BPH occurs in middle or older age men, when estrogen levels increase, while testosterone levels decrease. In France, BPH is treated with testosterone supplementation.

Testosterone supplementation appears safe, when limited to restoring testosterone to normal physiologic levels. Newer transdermal testosterone creams and gels bypass the gastrointestinal system, and more closely duplicate natural daily fluctuation of hormone levels, than weekly or monthly injections.



Phenylalanine is one of the eight essential amino acids and a precursor for phenyl ethylamine (PEA). PEA deficiency can produce depressions, which are unresponsive to tryptophan, 5-HTP, tyrosine, SAME or prescription antidepressants. High urinary PEA levels have been found in schizophrenics, while low levels have been found in depressed patients. Phenylalanine also is a tyrosine precursor, and tyrosine is a precursor to thyronine (thyroid hormone component) and dopamine. Phenylalanine may cause increased blood pressure, or pulse and should not be taken if you use monoamine oxidase inhibitors. (Beckmann, H., et. al., 1979) compared DL-Phenylalanine to imipramine, in a double blind study, of 40 depressed patients, for 30 days. No statistical difference was found, between the two treatments, on the Hamilton Depression Scale and the Bf-S self-rating questionnaire. Dosages used were 150-200 milligrams per day of DL-Phenylalanine or imipramine. Phenylalanine is available as D, L or DL Phenylalanine, but more commonly available as DL-Phenylalanine. D-Phenylalanine inhibits enkephalinase, which metabolizes enkephalins and endorphins and is used in some proprietary formulas to decrease pain and anxiety. L-Phenylalanine is the form used by the body to produce neurotransmitters and proteins. DL Phenylalanine is a mixture of D and L Phenylalanine.



Tyrosine is a naturally occurring amino acid and a precursor for the neurotransmitter dopamine and thyronine (substrate for thyroid hormone). Daniel Amen, MD, is a psychiatrist who has performed over 28,000 SPECT brain scans. Amen reports tyrosine is helpful for depressions, with reduced dopamine levels, and reduced prefrontal cortex blood flow. Reduced dopamine levels often occur in attention deficit disorder, and depressions where sleep, fatigue, lethargy and weight gain are increased. Low blood pressure and lower heart rates can also occur, with dopamine deficiency. Tyrosine may cause increased blood pressure, or pulse and should not be taken if you use monoamine oxidase inhibitors.

S-Adenosyl Methionine

S-Adenosyl Methionine

S-Adenosyl methionine (SAMe) occurs naturally in the body, crosses the blood-brain barrier and has proven effective treating depression and joint pain. It increases serotonin and dopamine levels and improves brain and nerve cell membrane fluidity.
S Adenosyl Methionine, SAMe, is produced in the body, from the amino acid methionine and Adenosyl Triphosphate, ATP. Conversion of methionine to SAMe is frequently impaired in cirrhosis and chronic liver disease.  Studies reveal  methionine supplementation does not increase SAMe level, in cirrhosis patients, since  SAMe synthetase enzyme levels are inadequate.  A double-blind, placebo-controlled, multicenter clinical trial performed in 220 patients with chronic liver disease revealed bilirubin decreased about 45 % with oral SAMe supplementation at 1,200 milligrams daily.

Almasio P, Bortolini M, Pagliaro L, Coltorti M Role of S-adenosyl-L-methionine in the treatment of intrahepatic cholestasis. Drugs. 1990;40 Suppl 3:111-23

S Adenosyl Methionine increased phosphatidyl choline levels in alcoholic liver disease.  Alcoholic liver disease reduces levels of phosphatidyl choline, a major component of cell membranes. SAMe produced increased glutathione levels in alcohol impaired livers and increased time till liver transplant was required.

Lieber, C. S-Adenosyl-L-methionine: its role in the treatment of liver disorders.  Am J Clin Nutr 2002;76(suppl):1183S–7S

Bell, K, et. al., (1994) conducted a double blind, placebo controlled study comparing SAMe to desipramine, in a 4 week trial with 26 depressed patients, at University of California, Irvine. SAMe produced improvement in 62 percent of patients, compared to 50 percent of patients treated with desipramine. Regardless of treatment, patients exhibiting a decrease of 50 percent or more in Hamilton Depression Scale, showed significant increases in plasma SAMe level. Salmaggi, P, et. al., (1993) found significantly greater improvement in depressed postmenopausal women treated with 1600 milligrams SAMe, than placebo. During the 30 day, double blind test of 80 women, between 45 and 59 years old, SAMe evidenced superiority by day 10 and side effects were mild. Amen, D. and Routh, L., (2003) have found SAMe useful for depression. Jacob Teitelbaum, MD (2003) recovered from chronic fatigue syndrome, and recommends 200 to 800 milligrams SAMe, twice per day, for depressed chronic fatigue and fibromyalgia sufferers, stating improvement can be expected in 10 days.

Peter’s Story

Peter’s Story – $600,000 of Rehabs Before Dr Sponaugle Stopped Peter’s Heroin Craving in Five Days

The holes toward the top of Peter’s brain scan are “dopamine holes.” Peter inherited a super COMT gene that causes a localized dopamine deficiency in the right prefrontal cortex, above the right eye. Peter “chased dopamine” through dopaminergic drugs to “feel normal.”

Peter was referred to Dr. Rick Sponaugle and Florida Detox® at age 28, he had a 16 year history of abusing Oxycontin, Heroin, Cocaine and Vodka – pretty much everyday of his life from age 12 years old. Peter’s family had spent $500,000 on “famous” addiction treatment centers in Arizona, California and New York. Yet, Peter had never abstained from drugs or alcohol more than four days since age 12 unless he was “locked up” in a rehab center.

Sadly, Peter was misdiagnosed by ill-knowledgeable psychiatrists working throughout America’s famous treatment centers. They continuously misdiagnosed Peter as a drug addict. Dr. Sponaugle diagnosed multiple brain chemistry issues in Peter that were causing his outrageous drug abuse.

Some were inherited like the localized dopamine deficiency seen on Peter’s brain scan, others he had acquired through various mechanisms. In all, Dr. Sponaugle treated over fifty biochemical deficiencies and imbalances which stopped Peter’s Heroin craving in just five days.

Peter also had multiple food allergies and a working diagnosis of Ulcerative Colitis from his NYU doctors. He had a severely toxic gut with leaky gut syndrome from the excessive steroids and antibiotics his New York doctors had prescribed for his Ulcertative Colitis. Their Big Pharma regimen had actually exacerbated Peter’s intestinal inflammation.

Steroids and antibiotics actually worsen LGS, leaky gut syndrome, they cause increased intestinal permeability and subsequently even more antibody attack on proteins that look like or mimic our body tissue, this we call autoimmunity. Both steroids and antibiotics cause intestinal overgrowth of Candida and Klebsiella, then their toxins cause multiple biochemical imbalances and deficiencies of brain chemicals.

Peter suffered severe serotonin deficiency and taurine deficiency because of his toxic and inflamed gut, deficiency of these two calming brain chemicals produces over activity in the brain’s  emotional center, thus, Peter used Heroin to calm excessive activity in his emotional center and for it’s dopamine stimulating effect on the dopamine deprived prefrontal cortex he inherited.

After Peter’s 16 years of drug abuse, multiple rehabs and abstinence of no more than four days, Dr. Rick Sponaugle used this type of scientific analysis and treatment to stop Peter’s drug and alcohol craving in just five days. When Dr Sponaugle explains the biochemical basis for addiction in his family workshop, it becomes obvious to the entire family that you can’t correct these biochemical causes of craving with counseling anymore than you can talk your way out of diabetes.

Steve Sponaugle, Research Director at Florida Detox®, recommended herbal medicines that Dr. Sponaugle used to heal Peter’s Ulcerative Colitis. Peter no longer needs to totally shut down his immune system with 60 mgs a day of Prednisone that was prescribed by his New York gastroenterologist. He no longer suffers symptoms of Ulcerative Colitis because his food alergies were properly diagnosed for the first time when he came to Florida Detox® &  Wellness Institute.

Peter’s Testimony:

It was Sunday April 27th, 2008, a day before my arrival at Florida Detox® and Wellness Institute. My girlfriend left me a week ago. The bank account was in the red and all the credit cards were maxed out. I had been flying high on a three-day rampage, devouring frightening amounts of crack and heroin. The house reeked of sour beer, stained with the horrid aroma of crack ash and scorched aluminum. Toxic sweat seeped from my pores, and pasted the clothes against my flesh. My face had been whittled to the bone and revealed a skull covered by a layer of sheer, grey, weathered skin. My eyes balls were glazed over and dry as rust. They stung from the stabbing strain of anxiety. My pupils swelled and stretched beyond their limits. My appearance alluded to the dark hollowness of my spiritless heart. My nerves imprisoned my ligaments and tendons while gnawing at the backs of my eyes. My stomach smothered itself and slurped on its own acids & bile. I franticly paced back and forth, peering out through the cracks in the blinds. A state of drug induced paranoid psychosis had crippled my mind. Periods of hallucinatory panic overwhelmed my reality. Thoughts screamed through my mind at decapitating speeds: Should I go? If I don’t go, what the hell am I going to do? …Die?????? I bet they’re no different from all the other places out there: “meetings, sponsor, work the 12 Steps, Blahh, Blahh, Blahh.” I‘ve heard that a million times, NO THANKS!!!! And they said painless detox? Yeah right! And my name is Santa who? Ohh…this sucks. I am f*&$ing hopeless. Once a junkie, now a junkie, die a junkie. I am such a useless idiot. I can’t do a damn thing right. I would %$# up free ice cream! Oh my god, ohh, I hate my life. I just want to be NORMAL… Is that too much to ask? Is it? I’ll give anything, just please, please help!!!




Today is Tuesday April 28th 2009, a date that I was sure I would never see. I entered Florida Detox® and Wellness Institute exactly one year ago today. As you can imagine I was in pretty bad shape, probably just days away from the unimaginable??? When I came to FL Detox and Wellness Institute I was ingesting 25+ bags of heroin a day and 60-80mg of methadone. And to top things off I had a scary crack/cocaine obsession. This so-called “habit,” as I used to call it, was costing over $400 to maintain. Before I go on any further, I would first like to tell you a little bit about my pilgrimage.


My addiction first sprouted at the age of 12. C’mon, you remember those times being young and foolish raiding your parent’s alcohol stash with friends? Yeah well, little did I know that I was embarking down a treacherous path of doom and despair? At the age of 13, my mother found the first bag of weed I had ever purchased (Looking back, I should of known right from the start). But anyway, after that incident my parents took DRASTIC measures. They immediately labeled me as an addict and made me go to an outpatient group therapy program. They also revoked my privileges and denied me of any interaction with friends outside of school. I attended group therapy twice a week and also met individually with a psychologist. My addiction by this point had progressed on to the likes of LSD, mushrooms, mescaline, dust and ecstasy. It continued to gather strength despite all efforts made by my parents. As things worsened and fear grew, my mother sought out a new outpatient group and counselor. This marked the beginning of a long and pricey route to a deeply disappointing truth: Today’s Addiction Treatment greatly lacks the discoveries and scientific advancements achieved by most medical sciences over the past century. In fact, it has hardly changed since Bill W. and Dr. Bob founded Alcoholics Anonymous 70 years ago.


*Today, you will pay anywhere from $30,000-$60,000 to go to the so-called “Top Inpatient Program in the nation” (FYI – they all make that claim). Okay, that’s ridiculous to begin with, but check this out; the real kicker is what you get for that hefty price tag. For $30,000–$60,000 you get to hideout for a month, and be told to work the following plan:


Your Keys to Recovery


(1) Go to AA Meetings


(2) Get a tenured Sponsor


(3) Work the 12 Steps


(4) Support System / Structured Schedule / Stay Connected

(5) Eliminate old People, Places, and Things / ID other triggers

(6) Healthy Diet/Nutrition/Meds / Daily Exercise Regimen

(7) Prayer, Meditation, and other techniques to De-Stress.

(8) Continuing Care (talk therapy, education etc.)

That summarizes today’s “BEST” approach in treating addiction. Notice anything ironic about this claim? Yeah, you guessed it…It has been the BEST approach for the last 70 years! Why is that? Who do you think manages most of the programs out there? Exactly, people whom either used AA to get sober, or who studied it in school. Sadly, it is all they know and god for bid they read about new studies and current trends???

Now you may say “wait a minute, what about all the other rehab programs such as Dual Diagnosis, Holistic, Christian, Individualized, Equine, Acupuncture, Spiritual, Luxury, Wilderness, Light, Continuing Care, Halfway House, Opiate Maintenance, and Behavioral?”

Okay, let me give you an example of what I mean. I’m sure you have noticed that many Rehabs rave about their unique ability to be able to tailor a treatment program based on the individuals needs? This is their idea of “uniquely tailored”: Johnny needs 3 meetings a day, and Jimmy needs 1 meeting a day. The bottom line is that all of those fancy names and scientific terms can be simplified down within those eight keys. I can assure you that 99% of all the rehabs out there will not offer you a different or better recovery plan. And if you have any doubts? By all means, go right ahead and get your checkbook out…

Before you go and blow all that money, let me finish telling you about the basis of my outrageous and preposterous claims. During my 16 years as an addict, I have seen 30+ therapists, ranging from your everyday psychologists, on to rare hypnotists, and even up to the lofty heights of the overpriced, top Psychiatric Addiction Directors. $305 gets you 15 maybe 20 minutes, in front of the top, Substance Abuse Psychiatrist at MUSC, a premier Medical Research University/Hospital in the Southeast. I, for instance, left his office with a prescription for an extremely low dose of Lexapro, and an appointment to return the next month. It was more than 6 months and $2,000 before he increased the dose of Lexapro to an acceptable therapeutic level!!!

I have been involved in over 15 different outpatient groups including IOP, (intensive outpatient) which would meet for four hours, five times a week. I have suffered in a handful of detox facilities and even tried various risky self-detox methods. One time I was using 800mg-960mg of oxycontin a day and tried doing a self-detox with suboxone…. Umm…. Yeaah, BIG Mistake!!!

Finally, last and by no means least, I have gone to five of the so-called “Top Inpatient Programs” in the nation including Pavillon, Clearbrook Lodge, and Daytop. My family wasted over $500,000 in therapy fees only to be told the same thing over and over, “Go to meetings, get a sponsor, eat healthy, exercise….” No Joke. Quite often, residential programs have patients like myself who come along and have a particularly difficult time withdrawing from the substances. The first three weeks of my stay at Pavillon (6 week program) were spent being bed ridden and suffering from the remnants of the detox. The rest of my stay was supposed to be focused on catching up on missed work. In actuality, it was spent struggling with severe biological imbalances that impacted everything from my energy levels and sleep cycles to my digestion and lack of focus. What made matters worse was the impending fate of the tsunami like cravings.

Do you know what I was told when I requested a medical revaluation? “Pete, you are an addict, who has been clean for only a short period of time.” “It is going to be difficult but you gotta remember that it will pass.” “Just follow the twelve steps, find a good home group, and make sure you get involved.” “Take it one day at a time!”

Sadly, I was shot full of dope two days later…

Now if you are anything like me, and do not want to spend your precious free time going to dark, dreary, mundane meetings, full of fearful people whom are, quite frankly, watching life pass them by, then you will agree:

“There has got to be a healthier, happier, more medically effective treatment to combat addiction???” I mean come on people! It’s the 21st century, the age of the Iphone, Barack Obama, and the Large Hadron Collider!


After 16 years of impending doom, the moment of truth had arrived…

Had all my years of deceitful sins finally caught up??? My girlfriend and I both knew that this was going to be the last chance for freedom. My mortality had arrived; I felt it piercing through, obliterating the self-righteous facade of my powerful, important and insanely obsessed ego. Astonishingly, my mortality revealed a young, fearful child who cried out for love and appreciation…

Suddenly, I jolted up out of the bed realizing I had been dreaming. I looked around slowly as I gained my focus bracing myself for the onslaught of the deafening vertigo and the paralyzing trembles that usually accompanied mornings without drugs??? A nurse knocked at the open door “Mr. Peter I have your breakfast, sir?” I motioned with my hand to come in. The smell of maple syrup made my mouth water, quickly alerting my starved stomach that food had arrived. I thought to myself “oh what the heck, let’s give it a shot.” Upon finishing breakfast, I decided to stand up. I slowly swung my legs over to the tile floor. I was positive that my blood pressure would fall out and ax down the shockingly sober presence that had graced me. Nope… Wow not even a slight head throb. Damn. My back was not even the slightest bit stiff. I knew what it was… they had not detoxed me yet. OK now it made sense. Nurse, Nurse, when uhm, are you going to do the detox procedure??? Pete, we already did it… You were out for a little over 24 hours. It went great, your labs look good, you are a little dehydrated but we are going to get some saline going here in a minute. If you want, you can go down and smoke a cigarette in the courtyard. Holy @#$%ing %$it!!!!!! I cannot believe it. I gotta pinch myself. Is this for real???


Well that was a year ago, and today I still have to pinch myself. I have been sober for one year. It may not sound like a lot to some of you, but when you consider the fact that over the past 16 years I had never been clean for more than four days…it is quite an achievement. Dr. Sponaugle and the rest of the team at Florida Detox® and Wellness Institute are fearless pioneers in the addiction business. Utilizing the latest research and technology available, they have devised a scientific treatment method that has an instant and profound effect on the entire mind, body, and soul. With extensive expertise in biological neuroscience, Dr. Sponaugle and the team are able to accomplish the results needed for sobriety in a very, very short time. What are the results? They will eliminate your cravings, stabilize your mood, sharpen your mind, and recharge your body so that you feel healthy, rested, motivated and alive. In addition to the drug and alcohol detox, they will cleanse your body of other harmful toxic impurities such as Candida. Following extensive lab tests they will replenish your body with the essential hormones, enzymes, vitamins and nutrients needed to sustain a healthy lifestyle. Drug and alcohol abuse takes a toll on the body and poses risks for many other illnesses. I, for example, had been diagnosed with Ulcerative Colitis. I saw four different specialists up and down the east coast and none of them could explain why I got it, how it could be treated or how it could be prevented. They said it was just “one of those things that had to run its course.” Florida Detox® and Wellness Institute did some testing of their own and sure enough, uncovered the fact that I had a severe reaction to turkey and eggs (FYI – I had eaten them daily). They also found that yeast was preventing my body from absorbing the vital nutrients needed to support healthy system processes. Finally, they discovered that I was missing an entire category of essential digestive bacteria in my large intestine. Today I can tell you that it has been one year without any recurrence’s of the colitis or any other digestive problems.

Look, the bottom line is that they cared about me. Because of them, I am ever so humbly grateful. Because of them, I know the Lord. Because of them, I have an extraordinary relationship with a beautiful woman. Because of them, I will marry her next spring. Because of them I will be a grateful, supportive, dependable, loving husband. Because of them, I was able to buy a home to begin our new life together in. Because of them, we are able to have kids. Because of them, I know I will be a wonderful & dedicated father. Because of them, I now enjoy real friendships. Because of them, I love and respect my family. Because of them, I have discovered and enjoy my true passions and talents. Because of them, I am comfortable & confident in my abilities and strengths. Because of them I am aware of, and am working on, my weaknesses. Because of them, I know who I am and where I am headed…

Because of Florida Detox® and Wellness Institute, I am alive!

How about you?

St. John’s Wort

St. John’s Wort

St. John’s Wort is a thoroughly researched herb, extensively prescribed by physicians in Europe for depression and anxiety. It has proven superior to placebo and at least as effective as amitriptyline, imipramine, maprotiline and paroxetine and usually produced fewer side effects. Insomnia and increased sun sensitivity sometimes increase with use of this herbal preparation. St. John’s Wort effect is similar to serotonin reuptake inhibitors although dopamine, interleukin and cortisol levels also appear to be affected. Some St. John’s Wort formulas contain Cadmium or Lead contamination and others do not contain the advertised or claimed amounts of hypericum. Unlike tryptophan, 5-hydroxytryptophan, phenylalanine, tyrosine and SAMe which naturally occur in the body, St. John’s Wort interacts with many medications. Zhou S, et. al. (2004) reports.

St John’s Wort decreased the blood concentrations of amitriptyline, cyclosporine, digoxin, fexofenadine, indinavir, methadone, midazolam, nevirapine, phenprocoumon, simvastatin, tacrolimus, theophylline and warfarin, whereas it did not alter the pharmacokinetics of carbamazepine, dextromethorphan, mycophenolic acid and pravastatin. St John’s Wort decreased the plasma concentration of the active metabolite SN-38 in cancer patients receiving irinotecan treatment. St John’s Wort did not alter the pharmacokinetics of tolbutamide, but increased the incidence of hypoglycaemia. Several cases have been reported that St John’s Wort decreased cyclosporine blood concentration leading to organ rejection. St John’s Wort caused breakthrough bleeding and unplanned pregnancies when used concomitantly with oral contraceptives. It also caused serotonin syndrome when co administered with selective serotonin-reuptake inhibitors (e.g. sertaline and paroxetine).

Some alcoholics and bipolar patients require treatment with an anticonvulsant, even after adequate magnesium levels are restored. Valproic acid and gabapentin have proven effective. In some cases anxiety will persist. Acamprosate is non addictive and offers new possibilities for reducing the anxiety of alcohol withdrawal and early abstinence. Insomnia is also common in the abstinent alcoholic. In some cases, tranquilizers may be needed temporarily to deal with insomnia, while melatonin, and amino acid supplementation are adequate for others.

Liver Detox

Liver Detoxification

Florida Detox® Alcohol Detox patients frequently arrive with elevated liver enzymes, alcoholic fatty liver or hepatitis. Typically, Florida Detox® regimens normalize elevated liver enzymes within one to two months.


The purpose of this article is to educate. This article does not constitute medical advice, diagnosis or treatment and is not a substitute for examination, testing, diagnosis and treatment, by a qualified, licensed health care professional.

Florida Detox® frequently detoxes opiate and alcohol patients, with elevated liver enzymes or hepatitis. Our alcohol and tranquilizer detoxification, at Florida Detox® is intravenous, with medication which eliminated by the kidneys, not the liver. Alcohol and tranquilizer detox does not require anesthesia. Patients with hemochromotosis are required to obtain treatment from another provider, for ferritin levels, exceeding lab reference ranges.
Florida Detox® uses milk thistle extract, alpha lipoic acid and selenium supplements to reduce liver enzymes in our alcohol patients, who often arrive with elevated liver enzymes. Typically, our regimen lowers elevated liver enzymes to normal, in one to two months.

This article is posted, with permission of Burton Berkson, MD and is available on the internet. He is at the Center for Integrative Medicine, in Las Cruces, NM and is a leading liver detoxification expert.

It is titled, Triple Antioxidant Therapy for Hepatitis C. Since this is already uploaded by someone else, onto the internet, I merely copied it, to post on this website. Maybe someone will copy this article to other sites, where it might receive more views-I believe Berkson’s protocol could help more people if it was more widely known.

Florida Detox® used this protocol with someone suffering from Hepatitis C and HIV. Two of three liver enzymes returned to normal, she regained about 16 pounds, her hair regrew and she suffers about half as much fatigue, since starting the protocol. She almost died, during prior interferon/ribavirin therapy with another physician. We also used colostrum, creatine, DGL, and IP6. She is caring for her children and grandson presently. (We did not treat her for chemical dependency)
Since this article was written, pharmaceutical interferon/ribavirin therapy effectiveness is reported to have increased to fifty percent of time. Herbal and neutraceutical therapies can be used together with the pharmaceuticals, although we recommend the more natural therapy be used in every case, since it is gentle, with almost no side effects.
Gary Null has some excellent articles discussing natural therapy for HIV, at his website, The Encyclopedia of Natural Medicine, by Murray and Pizzorno, has an excellent chapter, discussing Hepatitis C treatment. The website is another good source of information regarding Hepatitis C treatment.


As published in “Medizinishche Klinik”, a German medical journal.]


Background: There has been an increase in the number of adults seeking liver transplantation for hepatitis C in the last few years and the count is going up rapidly. There is no reliable and effective therapy for chronic hepatitis C since interferon and antivirals work no more than 30% of the time, and liver transplant surgery is uncertain and tentative over the long run. Since this article was written, pharmaceutical interferon/ribavirin therapy effectiveness is reported to have increased to fifty percent of time. This is because, ultimately, residual hepatitis C viremia infects the new liver. Furthermore, liver transplantation can be painful, disabling and extremely costly.

Treatment Program: The author describes a low cost and efficacious treatment program in 3 patients with cirrhosis, portal hypertension and esophageal varies secondary to chronic hepatitis C infection. This effective and conservative regimen combines 3 potent antioxidants (alpha-lipoic acid [thioctic acid], silymarin, and selenium] that possess antiviral, free radical quenching and immune boosting qualities.

Conclusion: There are no remarkably effective treatments for chronic hepatitis C in general use. Interferon and antiviral have less than a 30% response rate and because of the residual viremia, and a newly transplanted liver usually becomes infected again. The triple antioxidant combination of alpha-lipoic acid, silymarin and selenium was chosen for a conservative treatment of hepatitis C because these substances protect the liver from free radical damage, increase the levels of other fundamental antioxidants, and interfere with viral proliferation. The 3 patients presented in this paper followed the triple antioxidant program and recovered quickly and their laboratory values remarkably improved. Furthermore, liver transplantation was avoided and the patients are back at work, carrying out their normal activities, and feeling healthy. The author offers a more conservative approach to the treatment of hepatitis C that is exceedingly less expensive. One year of the triple antioxidant therapy described in this paper costs less than $2,000, as compared to more than $300,000 a year of liver transplant surgery. It appears reasonable, that prior to liver transplant surgery evaluation, or during the transplant evaluation process, the conservative triple antioxidant treatment approach should be considered. If there is a significant betterment in the patient’s condition, liver transplant surgery may be avoided.

Key Words: Hepatitis C treatment antioxidant alpha lipoic acid thioctic acid silymarin selenium

Background: More than 20 years ago, when the author was in medical and pathology training at 2 hospitals in Cleveland, Ohio, he was assigned to 6 critical patients who were suffering from acute hepatic necrosis secondary to hepatotoxic mushroom poisoning. He ordered thioctic acid (alpha-lipoic acid, ALA) from the National Institutes of Health and injected this antioxidant drug into the patients. In spite of their highly threatening condition the patients, as measured by laboratory values and clinical parameters, recovered quickly. Dr. Fred Bartter (then Chief of Hypertension and Endocrinology at the NIH) and the author were astounded by their recoveries and went on to treat many more mushroom poisoning patients. Over the years, the author continued to treat additional patients with other medical conditions using ALA, and observed similar results. The author has recently added silymarin and selenium to the regimen. In this paper he will discuss the use of this triple antioxidant regimen in the management of 3 chronic hepatitis C patients.
Patients and Method: The 3 basic antioxidants there were used in this report are alpha-lipoic acid (thioctic acid), silymarin and selenium (selenomethionine). The alpha-lipoic acid product was manufactured by Asta Medica at Frankfurt Am Main, Germany. The silymarin was a product distributed by NOW Foods of Bloomingdale, Illinois, and the selenium was encapsulated by Metabolic Maintenance Products Inc., of Sisters, Oregon.
The 3 patients were selected at random from a group of approximately 50 chronic hepatitis C charts at the Integrative Medical Center of New Mexico in Las Cruces. Each patient was maintained on a dose of 600 mg. of alpha-lipoic acid a day in 2 divided portions of 300 mg. each. The silymarin dose was 900 mg. per day in 3 divided portions of 300 mg. The selenomethionine dose was 400 mcg in 2 divided portions of 200 mcg. ( Enzymatic Therapy silymarin is complexed with phosphatidyl choline and is 8 times  more assimilable)

Because alpha-lipoic acid depletes some of the B vitamins, the patients were prescribed 2 B-100 capsules a day. In addition, each patient also took between 1,000 and 6,000 mg of vitamin C, 400 IU of vitamin E, and a mineral supplement. The patients were also requested to eat a daily diet that included at least 6 servings of fresh vegetables and fruits, only 4 oz or less of meat per meal, and 8 glasses of fresh water.
It was also suggested that the patients reduce their stress levels, and take part in an exercise program that included at least a 1-mile walk 3 times a week. The patients followed the nutritional supplement program carefully, however, it is not clearly known whether the other regimens were correctly followed.


Mrs. M.P. is a 57-year-old woman who acquired hepatitis C after a blood transfusion during surgery about 10 years ago. She did not eat a nutritious diet and did not live a very healthy lifestyle at that time. About 5 years ago, she became very fatigued and nauseous, and was diagnosed with non-A, non-B hepatitis. She was treated with conventional therapies and continued to degenerate into a poorer state of health. About 3 years ago she was diagnosed with chronic hepatitis C, cirrhosis, portal hypertension, esophageal varices, and thrombocytopenia, and treated with steroids and interferon. She did not improve. Her AFP (alpha-fetoprotein) level become elevated (16.1) and a mass was located in her liver. Mrs. M.P. was told that the mass was probably cancer and that there was no hope.
Mrs. M.P. presented at our office last year appearing fatigued, weak, pale, and her abdomen was grossly enlarged. The abdominal distention was due to ascites. She was administered oral furosamide (40 mg) and potassium chloride (10 meq) with a balanced diet and wholesome lifestyle. She lost almost 50 lb of fluid in 1 month. Mrs. M.P. was treated with 600 mg. of oral alpha-lipoic acid in 2 divided doses (300 mg each), 900 mg of silymarin in 3 divided doses (300 mg each) and 400 mg of selenium a day. A premium B complex vitamin was added to her regimen because alpha-lipoic acid depletes the body of thiamin, biotin and other B vitamins. Adequate amounts of vitamin C (2,000 mg), vitamin E (800 IU), Coenzyme Q10 (300 mg), and basic mineral supplements were also prescribed. Figures 1 and 2 track the favorable changes in her ALT levels and her AFP levels. Today, Mrs. M.P. is working 8 hours a day, feels healthy, looks good, and is not tired. She is free of the signs and symptoms of serious chronic hepatitis C infection.

Mrs. P.P. is a 49-year-old woman who was infected with hepatitis C following a blood transfusion prior to trauma surgery more than 10 years ago. During surgery, her spleen was excised because it was lacerated.
About 3 years ago, a liver biopsy was performed that showed moderate cirrhosis with active inflammation. As a result of this pathology, Mrs. P.P. went on to develop portal hypertension with esophageal varices. She never acquired thrombocytopenia because of the splenectomy, and did not show an elevated AFP. Mrs. P.P. was treated with interferon therapy without  satisfactory results. She was told that her condition was hopeless and that a liver transplant was her only option. Her health continued to decline and she presented at our office with fatigue, anxiety, and insomnia.
Mrs. P.P. was prescribed 600 mg. Of alpha-lipoic acid each day in 2 divided doses (300 mg each). To that, was added silymarin (900 mg/day) and selenium (400 ug/day). To combat the anxiety and insomnia, 0.5 of aprazolam was prescribed, as needed at bedtime. Mrs. P.P. was put on a balanced health and lifestyle program, and within 7 months regained her health. Figure 3 to 5 trace the favorable changes in her ALT levels, viral load and platelet levels. She is doing very well today and is working at an arduous job and playing at sports without any fatigue or other symptoms of serious disease.
Mrs. L.M. is a 35-year-old mother of 3 children who developed hepatitis C secondary to a blood transfusion during the birth of her baby girl 15 years ago. Three years ago she became ill and was diagnosed with cirrhosis of the liver, portal hypertension, and esophageal varices. As a result of the  portal hypertension, she developed splenomegaly and thrombocytopenia. Mrs. L.M.Ã’s hepatologist sent here to the university hospital for liver transplant evaluation. When she presented to our office, she was anxious, tired, and pale, and complained of constant pain in the regions of her liver and spleen. Mrs. l.’s fasting blood sugars were in the 300-mg/dc range. She did not have hyperglycemia prior to the hepatitis C infection. Mrs. L.M. decided that prior to the liver transplant surgery, she wanted to investigate a more conservative treatment regimen.
Mrs. L.M. was prescribed alpha-lipoic acid (600 mg./day), silymarin (900 mg./day) and selenium (400 ug) per day with other supportive supplements. She was encouraged to follow a healthy lifestyle program with a 2,000 calorie diabetes diet. Within 2 weeks she began to feel much better and recovered quickly. Her blood sugar fell into the normal range and the pain in her liver and spleen ended. She became energized and was able to do her normal work as a housewife. She returned to college the next semester earning a 3.8 grade point average (A). Figures 6 and 7 trace her favorable progress.



Alpha-lipoic acid (ALA) is a small organic molecule with a disulfide bond. It is a superb antioxidant that is soluble in both water and fat. ALA is an important coenzyme for the production of acetyl coenzyme A. Dihydrolipoic acid (DHLA), it’s reduced form, is an electron donor that recycles other fundamental antioxidants (vitamin C, vitamin E, and glutathione). ALA and DHJLA are superb free radical scavenger themselves because they neutralize peroxyl radicals [36], hydroxyl radicals [39] and singlet oxygen [38].
ALA is also a metal chelator that removes mercury from tissues [17], prevents calcium oxalate crystals (stones) from forming in the kidneys [21, chelates copper [28], and removes arsenic [18].
Lately, there has been a great explosion in ALA research. The lipoic acid/dihydrolipoic acid redox couple inhibits viral replication by stabilizing the NFK beta transcription factor [4], blocks the development of cataracts [24], protects the kidneys form aminoglycoside damage [35], insulates the pancreatic islet cells form inflammatory assault [7], inhibits thymocyte apoptosis , and stimulates the production of helper T cells [15]. In addition, the toxic side effects of cancer chemotherapy can be attenuated with the use of ALA [5] and it protects bone marrow from free radical damage secondary to ionizing radiation. [33].
Numerous other studies show that ALA is useful for the treatment of diabetes mellitus and syndrome X because it increased cellular glucose utilization [19]. And significantly reduces insulin resistance [12,20].
Diabetic neuropathy originates from a decrease in blood flow to various organs. This results in an accumulation of free radicals that can destroy nerve function. In one study, ALA brought about a significant reduction of neuropathic symptoms in 23 patients [46]. This was accomplished by abolishing the products of lipid peroxidation and increasing the entrance of glucose into the cell [27].
Due to ALA’s lipophilic characteristics, it can cross the blood-brain barrier quite easily and can scavenge free radical toxins in the central nervous system. Both ALA and DHLA protect animals from neuronal death following laboratory-induced cerebral ischemia and reperfusion experiments [9,16,32]. This effect is explained by the fact the ALA greatly increased glutathione levels in nervous tissue, thus protecting the nerves from the toxic products of oxidation.
For many years, ALA was used as a treatment for liver disease. As of yet, however, there are not many papers on this subject, and some studies used entirely sub-therapeutic dose [25].
Ethyl alcohol (ETOH) damages the liver by several mechanisms that ultimately lead to the proliferation of innumerable free radicals. These toxins damage the cell membranes by lipid peroxidation. It has been reported the ALA lowers the levels of ETOH metabolic breakdown products, and in consideration of this ALA may be an effective treatment for alcohol induced hepatitis, early cirrhosis, and alcoholic coma [23, 37].
In the late 1960’s and 1970’s, there were several studies describing the successful treatment of hepatotoxic mushroom poisoning with intravenous ALA [22, 47]. National Institutes of Health studies reported the survival of 73 out of 79 seriously poisoned patients [3, 6]. In American, interest in the use of ALA for hepatotoxic mushroom poisoning, and liver disease in general, was in the main lost, because of the growing fascination with liver transplantation as a proposed “standard of care” treatment for serious liver disease.


Silymarin is the mixed extract of the milk thistle plant (sylibum marianum) and has been used for hundreds of years as a treatment for liver disease. In the late 1960s and 1970s it was extensively used for serious hepatotoxic mushroom poisoning with excellent results [43]. It has been demonstrated to be a proficient antioxidant, protecting the liver by neutralizing dangerous hydroxyl radicals, superoxide ions and hypochloric acid. In this way silymarin neutralized the toxins that destroy the cellular membrane systems and the hepatocyte’s genetic material [10, 26, 41]. Silymarin, like ALA, increases cellular glutathione levels and decreases tumor promoter activity {1, 30].
Human viral hepatitis studies with silymarin demonstrate quicker normalization of liver enzymes, expeditious reduction of bilirubin levels, and shorter hospital stays [31]. In addition, silymarin has been shown to be an effective antidote for toluene and xylene toxicity, and drug overdoes [14, 29, 40]. Alcoholic and other chronic liver disease patients lowered their liver enzymes, decreased their levels of procollagen III, and improved the histology of their livers with daily oral administration of silymarin [2,13, 34]. Taking this intelligent reasoning into account, silymarin offers another effective treatment choice for serious liver disease.


Selenium (Se) is an essential metal that is required for normal antioxidant metabolism, reproduction and thyroid function. It is also an important coenzyme for the glutathione peroxidase detoxification system. Because of this, selenium neutralized peroxides that proliferate under oxidative stress and consequently protects cell membranes from free radical damage.
Selenium often combines with amino acids and forms selenoproteins. Viruses might benefit from being directly involved in this selenoprotein encoding process by monitoring selenium levels in the cell. Consequently, this viral behavior could act as a barometer for increasing or decreasing viral reproduction. If cellular selenoprotein levels fall, the virus might become more active and produce more viruses that attack new cells. If selenoprotein levels rise, the virus may remain in a dormant state for longer periods of time or remain permanently dormant.
Research papers have reported that RNA viruses, including hepatitis C virus, encode selenium-dependent glutathione peroxidase genes. In view of this concept, it is entirely possible that a specific viral gene could generate a selenium shortage in the host. And in this way, a selenium deficiency could stimulate viral proliferation and thus promote the progression of hepatitis C. To continue, in that case, the addition of selenium might act as a “birth control pill” for the virus and thus show down it’s reproduction mechanisms. According to several investigators this could give the immune system a chance to control the hepatitis C or HIV disease process [42,45]. EMPHASIS ADDED


There are no remarkably effective treatments for chronic hepatitis C in general use. Interferon and antivirals have less than a 30% response rate and liver transplantation is uncertain and tentative. Since this article was written, pharmaceutical interferon/ribavirin therapy effectiveness is reported to have increased to fifty percent of time. This is partially due to the residual viremia; the newly transplanted liver ultimately becomes infected again [44].   

S -Adenosyl Methionine can reduce elevated Bilirubin

S Adenosyl Methionine, SAMe, is produced in the body, from the amino acid methionine and Adenosyl Triphosphate, ATP. Conversion of methionine to SAMe is frequently impaired in cirrhosis and chronic liver disease.  Studies reveal  methionine supplementation does not increase SAMe level, in cirrhosis patients, since  SAMe synthetase enzyme levels are inadequate.  A double-blind, placebo-controlled, multicenter clinical trial performed in 220 patients with chronic liver disease revealed bilirubin decreased about 45 % with oral SAMe supplementation at 1,200 milligrams daily. Almasio P, Bortolini M, Pagliaro L, Coltorti M Role of S-adenosyl-L-methionine in the treatment of intrahepatic cholestasis. Drugs. 1990;40 Suppl 3:111-23

S Adenosyl Methionine increased phosphatidyl choline levels in alcoholic liver disease.  Alcoholic liver disease reduces levels of phosphatidyl choline, a major component of cell membranes. SAMe produced increased glutathione levels in alcohol impaired livers and increased time till liver transplant was required.   Lieber, C. S-Adenosyl-L-methionine: its role in the treatment of liver disorders.  Am J Clin Nutr 2002;76(suppl):1183S–7S


Leiber reports medium–chain triglycerides (MCTs) significantly reduce alcoholic fatty liver.  The main source of medium chain triglycerides, in the United States, is coconut milk or oil.


Hepatitis C positive patients are more likely to suffer hypothyroidism and benefit from thyroid hormone replacement. Hypothyroidism was observed in 13% of subjects with hepatitis C virus, compared with 3-5% of uninfected control subjects. In addition, the HCV patients had higher levels of anti-thyroid antibodies, markers of an autoimmune response.

En Espanol, Aqui

En Espanol, aqui

Florida Detox® ha marcado nuevos rumbos en un Desintoxificacion de alcohól en un metodo indoloro! La Desintoxificacion de alcohól no debería ser doloroso. Nuestros pacientes manifiestan que nuestra Desintoxificacion del alcohól impide que 98 % de los síntomas de retraimiento, que se han manifestado en otros programas de Desintoxificacion, esten presentes.

Nuestra Desintoxificacion médica para alcohol consta de 5-7 dias en Helen Ellis Memorial Hospital. Helen Ellis Memorial Hospital recientemente ha sido calificada, de acuerdo a los Rangos de Salud, en el 5% de superioridad, de los demas hospitales a escala nacional, en seguridad y control de calidad. Usamos medicamentos intravenosos que son más efectivos en el impedimento de los síntomas de retraimiento del alcohol. Nuestros pacientes NO EXPERIMENTAN ansiedad, espasmos musculares, náusea, vomito, diarrea, hipertension, pequeños temblores o alucinaciones. Durante la estadía del hospital los pacientes experimentan terapia de rejuvenecimiento con nutrientes y vitaminas intravenosas facilitando su transición inicial en la recuperación y bienestar general.

Nuestro programa de tratamiento de tres meses para alcohólicos provee de un acercamiento integrado hacia un tratamiento integral y el bienestar general. Los pacientes son evaluados para asuntos bioquimicos a nivel cerebral (psicológicos), desequilibrios que se manifiestan como deficiencia de atencion, ansiedad, depresión, insomnio, deficiencias nutritivas, desequilibrio hormonal, alergias ocultas a alimentos, hipoglucemia y crecimiento excesivo fungoideo a nivel systemico. Este tratamiento integral se acerca a la causa principal que produce el deseo al alcohol y da como resultado una prevención mas efectiva de reincidencia. La terapia de conversación, los 12 pasos, y el asesoramiento spiritual, son más efectivos una vez que estos asuntos sean tratados apropiadamente.

Después de completar una Desintoxificacion del alcohol segura y compasiva en un ambiente médico de calidad superior, que en el de un centro común del rehabilitacion, los médicos de Florida Detox® comenzarán a diagnosticar y tratar sus factores bioquímicos genéticos (ancestrales), que usualmente evita el deseo al alcohol. El asesoramiento espiritual es de vital importancia para asegurar la sanacion de heridas emocionales y el dolor del alma. Creemos fuertemente en la combinación de la ciencia moderna y el poder ungido de Dios para curar.

La Dependencia Alcohólica

El alcoholismo, también conocido como “la dependencia al alcohol,” es una enfermedad que incluye cuatro síntomas: El deseo: Una necesidad fuerte, o una compulsión, a beber. La pérdida de control: La incapacidad para limitarse a beber en cualquier ocasión dada.

La adicción física: Los síntomas de retraimiento, como náusea, transpiracion, estado tembloroso, y ansiedad, ocurren cuando se cesa el consumo de alcohol, después de un período prolongado de consumir alcohol La tolerancia: La necesidad para beber mayores cantidades de alcohol para “sentirse elevado”.

Las personas que no son alcohólicas algunas veces no comprenden por qué un alcohólico simplemente no puede huzar “un poco de fuerza de voluntad” y dejar de beber. Sin embargo, el alcoholismo tiene poco que ver con fuerza de voluntad. Los alcohólicos están en el agarre de una poderosa necesidad incontrolable “antojo,” para cosumir alcohol que pasa sobre la disposición de su habilidad. Aunque algunas personas pueden culminar su alcoholismo sin ayuda, la mayor parte de alcohólicos necesitan asistencia. Con tratamiento y soporte, muchos individuos pueden dejar de beber y reconstruir sus vidas.

Varias personas se preguntan por qué algunos individuos pueden usar alcohol sin problemas pero otros no lo pueden hacer. Una razón importante tiene que ver con la genética. Los científicos han encontrado que tener a un miembro familiar alcohólico incrementa mas las probabilidades de desarrollar alcoholismo, que si usted elige tomarse unas copas.

La fuente: Department de Salud y los Servicios Humanos de Estados Unidos. El Instituto Nacional de Abuso de Alcohol y el Alcoholismo. (2001, 1 de Enero). El alcoholismo: Trayendo los Hechos (NIH Publication No. 96-4153) Brochure . Washington, CD: U.S. Government Printing Office.

La genética

Evidencias considerable indica que la mayoría de alcohólicos geneticamente heredan fuertes propensiones hacia la adicción alcohólica. Schuckit, M., et. al.,(1972) estudiaron 133 Hijos de alcohólicos, adoptados y criados por padres no alcohólicos. Los 133 hijos de alcohólicos, criados por padres no alcohólicos, tuvieron una probabilidad tres veces meyor de ser alcohólicos, que los niños de padres no alcohólicos. También se ha encontrado actividad eléctrica anormal , en jóvenes, hijos de alcohólicos. Las hondas cerebrales P300 han sido frecuentemente estudiadas, en respuesta a estímulos suaves. Las hondas cerebrales P300 se encuentran suprimidas o ausentes en alcohólicos y sus hijos, que no consumen alcohol. Berman, et al. (1993) reporto que las medidas de la hondas P300 predijeron el uso de alcohol y drogas 4 años más tarde, (a la edad de 16) en un grupo de 36 niños pre-adolescentes.

Las diferencias genéticas también hace que el alcohol afecte a los alcohólicos en una formas diferente.La Dopamina posiblemente es la sustancia más adictiva del mundo y hace que la cocaína y la nicotina sean especialmente adictivas. La Dopamina crea sentimientos apacibles y se aumenta, durante el sexo o cuándo nos enamoramos. El alcohol eleva la dopamine, pero los niveles se reducenden durante el retiro alcohólico. Blum, kilobyte et al., (1996) investigaron el gene del receptor neurotransmissor tipo 2 (D2) de la dopamina y encontraron diferencias entre alcohólicos y no alcohólico. De 35 alcohólicos estudiados, 69 % presentaron el alelo A1, mientras que sólo un 20 por ciento de 35 no alcohólicos presentaron el mismo alelo A1. Estas variaciones genéticas determinan el número de receptores de Neurotransmissores D2. “Nunca he encontrado a un paciente que quiere ser adicto.” Están adictos a un enorme costo personal y social, pero algo fundamental ha cambiado en su materia gris a fin de que las motivaciones que normalmente motivan a otros, son desordenados o inhibidos por las drogas, ” dijo Nora D. Volkow, Maryland, Director de Escena del Instituto Nacional en el Consumo Ilegal de Drogas. . .

Dr. Volkow describió su estudio de 20 abusadores de cocaine hospitalizados que presentaron notable disminucion en las reducciones de receptores de dopamine D2, comparado con 23 controles. La disminución de receptores fue incluso aparente cuatro meses después de la descontinuación de la cocaína. Estas conclusiones prolongaron los resultados de otros estudios sobre reducciones del receptor de dopamina D2 en abusadores actuales de la cocaína y tambien mostro reducciones en receptores D2 en otras adicciones tales como el alcoholismo, heroína, y methamphetamine. “Este estudio documentó por primera vez los cambios bioquímicos en el cerebro de personas adictas, y resalto que las reducciones en los receptores de dopamina D2 y es muy duradero,”dijo ella. Los adictos tienen menos receptores de dopamina D2 en varias regiones de su materia gris que los controles. Durante liberación, la dopamina es casi inmediatamente extraida del synapsis por el transportador del dopamina, y así tiene una oportunidad muy limitada para alcanzar el receptor. Menos receptores para la misma cantidad de dopamina menos dopamina “recibida” y consecuentemente menos dopamina produciendo señales.

Contener más receptores de dopamina D2 en el cerebro le puede conferir como un beneficio protector en contra de la vulnerabilidad a la adicción, Dr. Notó Volkow. Ella y sus colegas han medido el número de receptores de dopamina D2 en un grupo de pacientes no addictos, luego les administro methylphenidato intravenoso. Los sujetos que describieron su experiencia como apacible tuvieron significativamente menos receptores de dopamine D2 que los que describieron una experiencia poco apacible. Al liberar mas Dopamina se provocó una experiencia antipática porque fomentó niveles del dopamine “sobre el umbral que sería considerado reforzador”. La experiencia fue demasiado intenso y tan desagradable que estos sujetos se negaron volver para un estudio de seguimiento, Dr. Volkow reportó .”

Yasgur, B. El Neurologic se arraiga de Adicción. NeuroPsychiatry Reviews. Vol. 5, No. 5; Julio del 2004

Las tomografías PET del cerebro revelan diferencias químicas en el cerebro entre adictos y las imágenes normales de los no addictos. Los imagenes normales en la columna inferior provienen de pacientes no addictos; Las imágenes anormales en la fila superior provienen de pacientes con desórdenes de adicción. Las esconografias PET del abusador de cocaína, el alcohólico, y el paciente obeso con función de adicción de comida, presento niveles reducidos de receptores de dopamine (las moléculas que transmiten le dan placer a las señales en el cerebro). Los niveles bajos de receptores de dopamine sugieren un “sistema” bioquímico con poca estimulacion de “recompensa” en el cerebro. La escanografia PET del fumador de cigarrillo con adicción a la nicotina muestra niveles inferiores de monoamina oxidasa (MAO), una enzima del cerebro que regula niveles de la dopamina. Los investigadores de reglas de codificación básica investigan terapias farmacéuticas para reprimir o aliviar comportamientos adictivos.

Algunos alcohólicos tienen niveles más altos de gen typo II para Alcohol dehydrogenasa, permitiendole a ellos una habilidad aumentada para la descomposicion del alcohol. Esto también está genéticamente determinado. Estos individuos comprometen aproximadamente 50 % de la población alcohólica y pueden metabolizar alcohol cerca de 40 % más rápido que otros alcohólicos y no alcohólicos. Los asiáticos tienden a contener menos de la enzima alcohól dehydrogenasa que abate el alcohol y tienden a tener intensas reacciones alergicas anaphylacticas al consumir alcohol parecido a las reacciones que ocurre cuando las personas beben durante el uso de antabuse (disulfuram). Los asiáticos experimentan un bolo de histamina, que a menudo es acompañada por la constricción de los bronquios, incomodidad intensa y dolores de cabeza. Consecuentemente, la tasa de alcoholismo es muy baja en la población asiática. En contraste, los Americanos Nativos tienen tasas de alcoholismo tan alto como el 80 por ciento. En algunos casos los americanos nativos sólo han tenido aproximadamente 200 años de exposición al alcohol, con poca oportunidad para que el alcoholismo se elimine de su mapa genetico. En el contraste, algunas poblaciones mediterráneas han tenido aproximadamente 7000 años de exposición al alcohol y tienden a tener tasas de alcoholismo muy bajas ya sean egipcios, españoles, franceses, italianos, griegos, turcos, sirios, libaneses, israelís, libios, o Morroccan. Esto tiene más que ver con la genética que con la religión, dado a que las religiones que se representaron alrededor del Mediterráneo incluyen la Cristiandad, judaísmo, y mahometismo. Esperanzadamente, los Americanos Nativos eventualmente engendrarán la abolicion de alcoholismo en su piscina del genes, pero hasta entonces, necesitamos más tratamientos efectivos. El primer paso de la descomposicion de alcohól en el cuerpo humano, es la transformación a acetaldehído, por la enzima alcohól dehydrogenasa. El acetaldehído es esencialmente una molécula de formaldehyde con un grupo de metilo (CH3) aderido, y es tóxico para cualquier tejido del cuerpo humano. Obviamente tiene un efecto prejudicial a nivel cerebral. Algunos alcohólicos heredan niveles cerca del 40 por ciento mas altos de la enzima alcohól dehydrogenasa que el resto del mundo, y descomponen el alcohol a acetaldehído, más rápido que otros bebedores. Desafortunadamente el hígado no puede descomponer la producción aumentada de acetaldehído mas eficazmente que cualquier otro bebedor, y los niveles aumentados de acetaldehído se concentran a nivel corporal.

Parte de la acumulación excedente de acetaldehído, que no es descompuesto rápidamente, a como se produjo, se combina con serotonin y dopamine, forman moléculas parecidas a opiodes denominadas tetrahydroisoquinolones, (THIQ). THIQ se une al receptor opiacio mu. Estos bebedores individuales, diferentes a los demas, se adictan mas intensamente al alcohol por los efectos producidos similares a los de los opiodes, secundario de el THIQ, que reducen ansiedad y dolor. La reducción resultante de ansiedad perpetúa adicción alcohólica. Cuando este individuo vuelve a la sobriedad, no sólo desean el narcótico (opiode), si no que también estan depletos de serotonin y dopamine, lo cual induce la depresión. Naltrexone es una droga de receta, que se usa para saturar el receptor opiode. Se ha encontrado que en aproximadamente 50 por ciento de alcohólicos reduce o elimina los deseos al alcohol, aunque comúnmente es usado para tratar adicción opiode.

En vista de estos hechos, es apropiado no mirar al alcohólico con compasión y desprecio. Desafortunadamente, mucha de la población no comprende estos hechos y aun tartan los alcohólicos desdeñosamente.

El Retraimiento Alcohólico

Muchos alcohólicos crónicos se convierten en bebedores de mantenimiento, bebiendo diariamente, para evitar síntomas de retiro. Los síntomas de retiro pueden incluir desasosiego de ansiedad, irritabilidad, nerviosismo, debilidad, insomnio, pérdida de apetito, patologias gastrointestinal, hipertension, pequeños temblores, convulciones y alucinaciones. Desafortunadamente, la repeticion del retiro alcohólico no medicamentado puede producir un exacervacion de pequeños temblores y depresión llamada “encendimiento”(kindling). La repeticion del retiro alcohólico y su reincidencia pueden ser más dañinos para la salud del alcohólico, que bebiendo de manera continuada como mantenimiento. Robert Post, en los Institutos Nacionales de Salud, encontor que el encendimiento también puede ocurrir con la precencia de epilepsia y depresión. Muchos programas alcohólicos tradicionales de desintoxicación impiden sólo las convulciones de gran mal. Estos programas a menudo permiten las náusea, el malestar gástrico, la ansiedad, el insomnio, el nerviosismo, los pequeños temblores, el pulso acelerado y la hipertension antihigiénica continuar, durante la desintoxicación. Los estudios PET (tomografía) del cerebro prueban que solo impidiendo la precencia de convulciones de gran mal, durante la desintoxicación alcohólica, permite el aumento de potenciales neurotóxicos de la membrana y de su voltaje que produce daños neuronales, y los daños son permanentes. Permitir que ocurra daño cerebral permanente durante la desintoxicación alcohólica es cruel e innecesario. ¿Por qué es esto admitido? El dominio de la anestesia a nivel experta elimina cualquier temblore o pequeños espasmos durante la desintoxicación. Dr. Sponaugle es un Anestesiólogo Certificado por la Junta Directiva de Anestesiologia y ha perfeccionado una técnica que impide estos síntomas desagradables y peligrosos.

En muchos alcohólicos, la severidad de los sintomas de retraimiento incrementan después de episodios repetidos de retiro del consumo de alcohol. Esta exacerbación puede ser atribuible al proceso de encendimiento. El encendimiento es un fenómeno en el cual un débil estímulo eléctrico o químico, lo cual inicialmente no causa respuestas conductistas abiertas, da como resultado la apariencia de efectos conductistas, como las convulciones, cuándo es administrada repetidamente. La prueba clínica y experimental apoyan la existencia de un mecanismo de encendimiento durante el retiro alcohólico. Los síntomas de retiro, como convulsiones, resultan de desequilibrios neuroquimicos en el cerebro de alcohólicos que repentinamente reduce o cesan su consumo de alcohol. Estos desequilibrios pueden ser exacerbados después de experimentar repetidos episodios de retiro al alcohol. La existencia de encendimiento (kindling) durante el retraimiento de alcohol sugiere que aun pacientes experimentando un retiro leve debería ser tratados agresivamente para impedir el incremento en la severidad de episodios subsiguientes de retraimiento. El encendimiento (kindling) también puede contribuir al riesgo de reincidencia de recaida de un paciente y al daño cerebral y al deterioro cognitivo. Becker, hidrógeno, (1998), p 25

Mientras el alcohólico es sedado con alcohol, el cuerpo humano sobre regula neurotransmitters excitativos incluyendo la acetylcholina, epinephrina, y glutamato. Al mismo tiempo, los mecanismos de información retroactiva bajo regulan el GABA (ácido gamma-aminobutyrico), el cual es el neurotransmitter más inhibitorio y abundante en el cerebro. El magnesio también es gravemente depletado por el alcohol. El magnesio es necesario para relajar músculos y es usado en el tratamieno hospitalario del delírium tremens, el cuál puede ser fatal, si no es tratado. Durante el retiro alcohólico, pequeños temblores y convulciones pueden ocurrir, dado a que los neurotransmitters excitativos como acetylcholine, glutamate y epinephrine estan elevados, mientras que el neurotransmitter inhibitorio GABA y el magnesio en suero estan disminuidos. Gabapentin, Topiramato y Clonidina son algunos de los medicamentos usados para reducir severidad de pequeños temblores. Gabapentin es un anticonvulsivante, con un parentesco estrecho al neurotransmitter inhibitorio, GABA. Gabapentin puede ser usado, cuando hay precencia de daño hepatico, dado a que es eliminado, por la orina, y no metabolizado por el hígado. Topiramato es un anticonvulsant moderno que también es poco adictivo. La Clonidina (Catapres) es usado para reducir la presión sanguínea y el latido rápido del corazon durante el retraimiento al alcohól.

Los medicamentos que pueden disminuir el deseo (antojo) y incrementar la abstinencia incluyen el Acamprosate, Naltrexone y Topiramato. Acamprosate, fue recientemente aprobado por la Administracion de Alimentos Y Medicinas, y se ha usado mas de 10 años en Europa para el manejo de la ansiedad aumentada durante la abstinencia al alcohol, y ha mostrado incremento en la taza de sobriedad. Acamprosate parece reducir la liberación de glutamato en la union pre-synaptica. Acamprosate se puede usar, cuando hay presencia de daño hepatico, dado a que es eliminado, por la orina, y no metabolizado por el hígado. Naltrexone es un antagonista opiaceo y – se ha probado – que reduce los deseos (antojos) alcohólicos y las tasas de reincidencia. Se ha probado que Topiramate también incremento la taza de abstinencia y parecen aumentar niveles del GABA.

Alcohólicos Anónimos

Muchos alcohólicos son incapaces de “trabajar” (seguir) los 12 Grados de Alcohólicos Anónimos exitosamente, hasta que las deficiencias nutritivas y los daños fisiológicos, causado por alcohol, son corregidos. La supplementación nutritiva y el tratamiento médico pueden reducir o eliminar los deseos alcohólicos, la hipoglucemia, deficiencies de ácidos grasos esenciales , depresión, cambios bruscos del ánimo, ansiedad, insomnio, pequeños temblores, pérdida de memoria, hígado graso, gastritis, reflujo gastro-esophagico, mal-absorption, aumentó de alergias secundarias a la permeabilidad intestinal, alergias alimenticias, desequilibrios hormonales, y otros problemas que impiden a los alcoholicos controlar su vicio a la bebida.

Alcohólicos Anónimos y modelos de 12 Pasos continúan dominando la mayoría de programas de tratamiento para el alcohólico. Su dominancia se persive en un mensaje no escrito al público, ese programa de tratamiento de 12 Pasos para alcohólicos son los programas de tratamiento más efectivos para los alcohólicos. La literatura de Alcohólicos Anónimos ha sido traducida a muchos lenguajes y ahora muchas reunions, para no fumadores, están disponibles. El Libro Grande de Alcohólicos Anónimos fue publicado en 1939 y estan esencialmente inalterados desde entonces. Desafortunadamente, los Alcohólicos Anónimos han ignorado avances médicos, científicos y nutritivos, los cuáles van acelerando progresivamente.

Los programas nuevos para alcohólicos, que son basados en tratamiento nutricional o biológicos, producen tasas de sobriedad tan altas como del 70 al 80 por ciento, durante un año. La efectividad del tratamiento alcohólico nutritivo fue probada en los inicios de 1950 y documentado por Guenther, R. (1983) quien transmitió un estudio de 6 meses en un Centro Médico de la Gerencia de Veteranos, en Waco, Tejas, con 108 alcoholicos internados. Todos los pacientes participaron en reuniones de desintoxicación alcohólica y de educativas, reuniones de Alcohólicos Anónimos, consejeria en grupo y seguimiento del paciente no hospitalizado, después de un programa de 28 dias. El grupo experimental de nutricion participó en el mismo programa con el mismo grupo de planta, educacion de nutrición, suplementos nutritivos y un régimen más nutritivo. Las modificaciones dietéticas incluyeron germen de trigo y salvado con cada comida, pan del grano entero, bebidas descafeinadas, y substitutos de azúcar. La fruta no azucarada sirvió para postre, en lugar de pastel, pudín o fruta endulsada. Los bocadillos permitidos fueron frutos secos o granos como mani, queso, pan de multiples granos y mantequilla de maní. Seis meses después de la descarga hopitalaria, 81 por ciento del grupo experimental nutritivo no bebía, comparado con un 38 por ciento del grupo testigo. La probabilidad estadística de una diferencia en rangos de sobriedad de este gran valor en un grupo de este tamaño se calculó de ser dos casos en 1000. (P = 0.002) (Para lectores poco familiar con estadísticas, p = 0.002 quiere decir la probabilidad de una diferencia ocurriendo por casualidad es 2 en 1000. p = 0.05 significa 5 acasos en 100, o una probabilidad de 5 por ciento a que una diferencia ocurra por casualidad. Este estilo de notación de probabilidad será usado más adelante en este artículo.)

Si usted pasa bastante tiempo en grupos de Alcohólicos Anónimos, probablemente escucharás que sólo cerca de uno de cuatro participantes de Alcohólicos Anónimos están sobrios un año más tarde. Dórese, R., et. Al(1990).cito a Harrison y Hoffman (1988) quienes reportaron que el 73 por ciento de alcohólicos que asistieron a Alcoholics Anonymous semanalmente, durante al menos 6 meses estavan sobrios, pero el 30 a 50 por ciento de alcohólicos que inicialmente asistieron a los Alcohólicos Anónimos no terminaron el programa. Brown, R., et. al. (1990) cito a Hunt, W. et. al. (1971) quien encontró una tasa promedio de sobriedad de alcohol durante un año del 33 por ciento; Bill, C. (1965) quien encontró que 34.6 por ciento de miembros de Alcohólicos Anónimos fueron abstinentes, al final de un año, tambien Milkman y Sunderwirth (1987) que encontarón, “aproximadamente 75 % de todos los que intentan abstinencia al alcohól cambiaron sus hábitos entre tres a seis meses después de empezar un programa de recuperación”. Algunos grupos de Alcohólicos Anónimos dan la apariencia de estar interesados en posibilidades de tratamiento bioquímico. El grupo de Alcoholics Anonymous del Oeste de Baltimore, ha integrado el siguiente pasaje, por Abram Hoffer, Maryland, PhD, para su sitio Web.

El término vitamina B-3 fue introducido por mi amigo Bill W., co-fundador de Alcohólicos Anónimos, (Bill Wilson). Nos conocimos en Nueva York en 1960. Humphry Osmond y lo presentamos al concepto de Mega terapia vitaminaca. Describimos los resultados que habíamos visto con nuestros pacientes esquizofrénicos, algunos también alcohólicos. También le dijimos de muchas otras propiedades. Fue terapéutico para la arthritis, para algunos casos de senilidad, y disminuyo niveles de colesterol.

Bill estaba curioso sobre el tema y comenzó a tomar niacina, 3 g diariamente. Dentro de unas semanas la fatiga y la depression, que le habían plagado por años, culmino. Él se lo dio a 30 amigos cercanos en Alcohólicos Anónimos y los indujo a probarlo. Dentro de 6 meses él estaba convencido de que era muy útil para los alcohólicos. De los treinta, 10 fueron liberados de su ansiedad, tensión y depresión en un mes. Otros 10 se sanaron en dos meses. Él decidió que los términos químicos o médicos para esta vitamina no eran apropiados. Él quiso convencer a los miembros de Alcohólicos Anónimos, especialmente los doctores en Alcohólicos Anónimos, que esta era una adición útil para el tratamiento y necesitava un término que fuera más fácilmente popularizado. Él me preguntó los nombres que se habían usado. Le dije que fue originalmente conocido como vitamina B-3. Éste fue el término que Bill quiso. En su primer informe para los médicos en Alcohólicos Anónimos él le llamó “la Terapia de Vitamin B-3″. Miles de copias de este folleto extraordinario fueron distribuidas. Eventualmente el nombre regresó y hoy hasta las publicaciones médicas más conservadoras utilizan el término vitamina B-3.

Bill se volvió impopular con los miembros del Grupo de Consejo internacional de Alcohólicos Anónimos. Los miembros médicos, quienes habían sido escojidos por Bill, consideraron que él no tenia ningun motivo para cambiar el trataminto con vitaminas. También “se enteraron” que la vitamina B-3 no podría ser terapéutica como Bill habia encontrado que era. Por esta razón Bill le entrego la información a los miembros médicos de Alcohólicos Anónimos fuera de la Junta Directiva Nacional, distribuyendo tres de sus folletos asombrosos. Ahora no están fácilmente disponibles. ADVERTENCIA: Niacina, en gran escala o en mega-dosis, por períodos prolongados pueden producir toxicidad hepatica. La vitamina B-3, está disponible como Niacinamida, Inositol Hexaniacinato o Dinucleotido Hydrogeno Dinucleotido de Adenina Nicotinamidada, (NADH) las cuáles son más seguros para el hígado. Florida Detox® no recomienda supplementación en megadose de Vitamina B-3 para el alcoholismo, sin pruebas de laboratorio de las enzimas hepaticas. A pesar de la resistencia al cambio por parte de Alcohólicos Anónimos, la participación en Alcohólicos Anónimos le ofrece ventajas considerables al consumidor de alcohol. Los Alcohólicos Anónimos le ofrece a los alcohólicos un soporte emocional sin paralelo. Si el alcohólico diligentemente cultiva relaciones, en grupos de Alcohólicos Anónimos, tiende a unirse con personas compasivas que trabajan y duermen en un agenda de horarios variados. Suficientes relaciones potencialmente le podrían proveer acceso 24 horas al día, siete días a la semana a un compañero, o el soporte emocional nesesario durante una crisis. Los Alcohólicos Anónimos están extendidos geográficamente, proveyéndole compañeros y soporte al consumidor de alcohol, durante un viaje. Los alcohólicos asociados al grupo a menudo pueden captar cuándo alguien está en peligro de reincidencia, más temprano que consejeros no alcohólicos o consejeros profesionales de adicción que nunca fueron químicamente dependientes. Los alcohólicos tienden a tener memorias selectivas de sus experiencias durante el consumo de alcohol, olvidando los peores desastres, pérdidas, y devastaciónes, mientras recuerdan los momentos más lejanos de experiencias alcohólicas agradables. Escuchar experiencias de otros alcoholicos les recuerda la devastación y la destruccion que el alcohol les causó.

El quinto paso de Alcohólicos Anónimos implica revelar o compartir sus peores errores, sus defectos y sus fracasos con otra persona. Compartir estos secretos vergonzosos con una persona que no jusge, reduce la sensacion de verg�enza y compulsiones para perpetuar comportamientos indeseables.

El paso 12 de Alcohólicos Anónimos puede ser lo más poderoso e implica compartir su fuerza, su esperanza y su recuperación con otras víctimas. Este paso es una paráfrases de la ley bíblica de sembrar y cosechar. Mientras el alcohólico siembra la recuperación en la vida de otra víctima, la recuperación fluye en la vida de alcohólicos. Un patrocinador de Alcohólicos Anónimos es un mentor que “patrocina” miembros con menos sobriedad, compartiendo las perspectivas y las experiencias ganadas al recuperarse. Estadísticamente, estos patrocinadores, tienen tasas mínimas de reincidencia, en Alcohólicos Anónimos. Algunas veces los patrocinadores han estado al borde de una reincidencia, cuando recibieron una llamada de auxilio, de alguien. Los Alcohólicos Anónimos tienen muchos esloganes (dichos) contagiosos, incluyendo, “usted no lo puede conservar (la recuperación), a menos que usted lo regale. Cuando los patrocinadores se percatan de que pueden transformar la peor experiencia de su vida en algo poderoso, para ayudar a otra víctima, su vida gana propósito y un significado nuevo. En Florida Detox®, muchos de nuestros pacientes recuperados escriben testimonios o se ofrecen a hablar con pacientes prospectivos, efectivamente, “trabajando” el 12avo paso.

El Tratamiento Alcohólico

Después de que la desintoxicación alcohólica ocurre, hay que anfatizar sobre asuntos, que posiblemente causo el alcohólico beber o efectos secundarios del alcoholismo, incluyendo el déficit de atención, la depresión, la ansiedad, las alergias a alimentos , el estrés pos traumático, la hipoglucemia, la deficiencia de vitaminas, minerales, o ácidos grasosas esenciales y el desequilibrio de hormonas.

Naltrexone fue originalmente aprobado en 1984 para tratar la adicción a heroína. En 1994, la Administracion de Alimentos Y Medicinas aprobó naltrexone para tratar alcoholismo. Los pacientes tratados con naltrexone y terapia no hospitalaria, tienen el doble de probabilidad para abstenerse a cualquier vicio de bebidas, o a recaer, que pacientes que sólo recibiendo terapia del paciente no hospitalizado. Además de naltrexone, las pruebas de investigaciónes sobre nuevos medicamentos son prometedoras para un éxito en el futuro. En un estudio de nalmefene, por ejemplo, se encontró que los pacientes alcohólicos que recibieron el medicamento fueron 58 por ciento menos probable para reincidir que pacientes que recibieron un La incidencia más alta de reincidencia en pacientes con dependencia del alcohol ocurre durante los primeros meses despues de la culminacion del consumo de alcohol. Nalmefene es un antagonista opiode moderno, como naltrexone, que no tiene actividad agonista y ningún potencial de abuso. Las ventajas adicionales incluyen mejor bioavailability, una vida media mas prolongada, y ninguna toxicidad hepatica basada en dosis. La posibilidad de reincidencia fue 2.4 veces mayores en placebo que aquellos pacientes que recibieron nalmefene. Otras conclusiones en los pacientes recibiendo nalmefene incluyeron periodos más cortos de recaidas, un mayor número de días hasta la primera reincidencia y menos bebidas consumidas en los días que una reincidencia ocurrió JEFFREY T. KIRCHNER, D.O.

Mason BJ, et al. Un estudio doble ciego, controlado con placebo de nalmefene oral para la dependencia del alcohol. Psiquiatría Arco del Gen 1999;El 56:719-24.

La Reducción Drástica de Nutrientes

Se ha encontrado que todas las deficiencias de vitamina B1, B2, B3, B5, B6, B12, ácido folic, y biotin – causan depresión, y el alcohol agota todo estas vitaminas. Alpert, J. y Fava, M. (1997) encontro que ente 15 a 38 por ciento de los pacientes deprimidos fueron deficientes en ácido folic. El alcohol agota todas las vitamina B, Vitamina A, Vitamina C, Vitamin D, Vitamin E, calcio, todo el magnesio, todo el cinc, todo el potasio y todo el selenio.

El Metabolismo de ácidos Grasoso Esenciales

El alcohol aumenta la depresión e inflamación alterando niveles de ácido grasos esencial y prostaglandinas. El alcohol agota el magnesio, cinc, y vitamina B6, interfiriendo con la enzima desaturadora delta 6. El desaturadora delta 6 es necesarios para convertir a los aceites vegetales Omega 3 al ácido eicosapentaenoico(EPA) y los ácidos grasos esenciales decosahexanoico (DHA) y los aceites vegetales Omega-6 a prostaglandinas inflamatorias(PGE2). Los aceites vegetales artificialmente hidrogenados, también se adjieren a los sitios activos de aturacion de la enzima desaturadora delta 6, impidiéndolo su funcion. Estas grasas hidrogenadas, previamente líquidas a temperatura ambiente, están alteradas por la adición de un hidrógeno adicional a los átomos de carbóno, transformándolos de no saturado a grasas saturadas. La hidrogenación aumenta durabilidad y spancibilidad de aceites vegetales, margarinas y mantequilla de maní. La hidrogenación también endersa curvaturas, en la molécula gruesa, alterando la configuración física. Estas moléculas alternadas, disminuye la permeabilidad de la membrana plasmática, interfiriendo con la liberación y recepción de neurotransmisores, nutrientes y transporte residual. Las grasas hidrogenadas deberían ser eliminadas del régimen.

ácidos Grasos Omega 3
Sin la enzima de desaturaicon delta 6, flax y otros aceites vegetales de Omega 3 no pueden ser transformados al anti-inflammatory Acido Eicosapentaenoic (AA). La produccion de DHA de EPA también requiere del desaturador delta 6. Barry Sears, PhD, explica cómo la EPA y DHA reduce ataques del corazón, infartos, y hipertensión, artritis, asma,enfisema,cáncer, infección, enfermedad de Alzheimer y depresión, en su mejor libro de venta, El Omega Rx Zone. DHA, es la grasa más abundante en el cerebro, la cuál es 60 por ciento grasa. En un studio de metodo doble ciego, con un placebo control, en Harvard Medical School, Stoll, A., et al.(1999), compararon la supplementación alta de aceite de pescado Omega 3 al aceite de oliva, en 30 pacientes con enfermedad bipolar. En el estudio de cuatro meses, los pacientes suplementados con aceite de pez, se presento mejor en casi cada resultado medido, especialmente el tiempo para reincidir la depresión o la manía. En cuatro meses, la reincidencia de depresión o manía fue 47 pecent inferior, en pacientes consumiendo altos niveles de aciete de pescado Omega 3. La dosis fue de 6.2 de ácido eicosapentaenoico (EPA) y 3.4 gramos de acido decosahexanoico (DHA) diariamente. Ocho sujetos en el estudio no estaban usando medicamento farmacéutico para enfermedad bipolar y estaban igualmente divididos entre el grupo experimental y el grupo testigo. Los cuatro sujetos no medicamentados tratados con aceite de pescado se mantuvieron libre de remisión considerablemente más tiempo que los sujetos no medicamentados del placebo. Estadisticamente, la probabilidad de que esta diferencia ocurra por casualidad, fue de 4 en 100. (P = 0.04).

ácidos Grasos Omega 6
Los niveles de los ácidos grasos del Omega 6 están alterados en la depresión y el alcoholismo. Afortunadamente, pueden ser suplementados para disminuir depresión. El aciete de omega 6 compromete más del aceite vegetal en el régimen americano e incluye soja, sésamo, maíz, aceite de girasol y alazor. Estos aceites son inicialmente convertidos a ácido de gama-linolenico o GLA, por la enzima desaturadora delta 6. Como comentamos previamente, el alcohol interfiere con la enzima desaturadora delta 6. Niveles de GLA estan disminuidos en pacientes deprimidós y alcohólicos. GLA es un precursor del ácido linolenico gama Dihomo (DGLA) que puede ser convertido a Acido Arachidonico (AA) o a prostaglandinas antiinflamatorias (PGE1). El AA es un precursor para varias prostaglandinas inflamatorias. Cuando menos AA es producidos del DGLA, más PGE1 antiinflamatorio prostaglandins puede ser producido del DGLA. El EPA disminuye la producción de (los AA) limitando la producción de la enzima desaturadora delta 5. Adams, P, et. al. (1996) encontro que el rango entre (AA)/EPA incremento con severidad en la depresión, en un estudio de 20 pacientes moderado a gravemente deprimidos. A medida que el AA disminuyen, la producción inflamatoria del prostaglandin decrece, mientras la producción anti-inflamatoria aumenta. Una de las prostaglandinas inflamatorias producidas de AA, es PGE2. (Lieb, J., et. al., 1983) midieron prostaglandina inflamatioria PGE2 en treinta pacientes deprimidos no hospitalizados y encontró que los niveles del PGE2 en plasma fueron elevados en 29 a 30 pacientes.Las prostaglandinas inflamatorias PGE1 pueden contrarrestar las PGE2 prostaglandinas inflamatorias PGE2, las cuales estaban elevadas en la depresión.

En resumen, los niveles crecientes del EPA pueden reducir la severidad de la depresión, por la suprecion de la producción inflamatoria de prostaglandin y el aumento de producción de prostaglandinas anti-inflamatoria. Supplementation con aceite de pescado puede aumentar niveles del EPA eficazmente y puede disminuir la depresión.

Supplementation con aceite de pescado en concentracion farmacéutica, puede sobre pasar eficazmente el deterioro de la enzima desaturadora deltas 6, sea por inducio genético o por consume de alcohol. Dado a que los pescados de agua fría contiene altos nivels de EPA y DHA, la produccion de la enzima desaturadora deltas 6 no es necesaria de los aceites vegetales. Los nuevos aceites de grados farmacéuticos están destilados para remover el mercurio, bifenilos policlorificados (PCBs) y otros contaminantes, los cuáles pueden bio-concentrarce en peses de mayor tamaño. Usualmente no ocurre eructos, desde que la destilación extraiga los sabores y olores “de dudoso origen”. El EPA y DHA son concentradas por la destilación, reduciendo la necesidad para tomar más de una cuchara al día. Los aceites son condimentados con limón, cal o naranja y realmente tienen un sobor agradables. El peligro de toxicidad de Vitamina D o sobredosis se elimina si el suplemento no contiene aceite de hígado o de bacalao. Cuando hay dolor de articulacion, asma, enfisema y otras condiciones inflamatorias las fórmulas con una concentración superior del EPA a DHA, puede usarse para antagonizar la inflamación.

La supplementación de GLA también puede reducer la depresión y consumo de alcohol. La Borraja, la grosella negra o los aceites de la prímula de noche puede usarse para fomentar niveles GLA. David Horrobin, (1985) trato varios alcoholicos en un studio controlado con un placebo con GLA, durante un año en Escocia. GLA redujo la dosis requeridas de tranquilizantes, síntomas de retiro, y deseos de consumir alcohol, más que en el grupo placebo. GLA mejoró la function hepatica rápidamente y aumentó abstinencia al alcohol durante un año.

La Hipoglucemia

Diferentes investigadores han encontrado que el 50 al 96 % de alcohólicos son hypoglycemicos. Poulos, J, et. al. (1979) administrado una prueba de tolerancia a la glucosa de 6 horas a 50 alcohólicos internados “mitad de tiempo en el hospital” y 50 alcohólicos no hospitalizados. Todos los 50 pacientes alcohólicos no hospitalizado, estaban hypoglycemicos, y 46 a 50 alcohólicos internados mitad del tiempo, estaban hypoglycemicos, con un diabético y 3 alcohólicos prediabéticos medio internados. Asi fuera un resultado del alcohol o si ellos trataban la hipoglucemia con alcohol, quizá sea irrelevante una vez que se convirticen en alcohólicos. Los síntomas de hipoglucemia en muchos casos son idénticos y es denominado síndrome del borracho seco en Alcohólicos Anónimos. La depresión, la irritabilidad, la ansiedad, el insomnio, la concentración deteriorada, el mareo, y el dolor de cabeza son síntomas comunes de la hipoglucemia. La hipoglucemia es evidente en grupos de Alcohólicos Anónimos. En las reuniones de Alcohólicos Anónimos, los clientes habituales comúnmente beben gaseosas durante sus vistas, y otros refrescos, los cuáles contienen hasta 10 cucharadas de azúcar o llenan una taza de café de icopor con una cuarta parte o medio de azúcar blanco y colmándola con café normal, lo cual aumentará liberación de insulina y de glicógeno para levantar azúcar en sangre rápidamente. F. sana, et. al. (1991), Midio la función mental de 67 pacientes, durante una prueba de tolerancia de glucosa de cinco horas. Los pacientes Hypoglycemicos requirieron significativamente (p = 0.0002) más tiempo para realizar sustracciones seriales de siete (problemas matematicos de resta), que pacientes no-hypoglycemicos.

” El quid del problema de alcoholismo es hipoglucemia ya sea que ocurra en la persona predispuesta por factores genéticos influenciando su crecimiento físico y emocional, o en el bebedor social que tiene agravando la función de carbohidratos, o en el alcohólico crónico en el Síndrome General de Adaptación (Hans Sleye, M.D.) estado de resistencia o fatiga, o en el alcohólico recuperado con sus quejas psíquicas y físicas. Aun en la persona normal, una hipoglucemia temporal se presenta como el cuadro clinico previo. (En 1968 Bill W., Fundador de A.A., pretextado con médicos en 48 páginaa, “la X” 81/2 “11″ folleto para usar en el régimen de hipoglucemia y las vitaminas, especialmente B3, en el tratamiento de alcoholismo. (Esto está agotado pero nosotros tenemos una copia que fotocopiamos de vez en cuando.) El Título ” la Terapia de Vitamina B-3; Un Segundo Communicado para los medicos de AA de Bill. W “. Febrero, 1968. Bill W. escribió, después de una descripción de la condición hypoglycemica, ” La víctima es alternativamente aserrada entre demasiada insulina y demasiada adrenalina. Inconscientemente, nosotros los alcohólicos intentamos curar estas condiciones primero con dulces, y luego con café. Los dulces levantan nuestra glucosa en sangre, y percevimos una mejoria. El café también nos da un estímulo temporal dado a que reduce el choke de la disminucion de glucosa en sangre. Exactamente por la vía equivocada, estamos inconscientemente tratando nuestra hipoglucemia. Si usted ya consume B-3 y Vitamina C, sume la disciplina dietética. Si usted tiene hipoglucemia de cualquier extensión, los dividendos están dispuestos a ser muy grandes “. La asociación de hipoglucemia, Julio /agosto IncBulletin #200 Sept.1996 Box 165, Ashton, Maryland La glucosa se deriva de tres fuentes: alimentos; síntesis (la produccion) en el cuerpo humano; Y la descomposicion de glicógeno, una forma de glucosa que el cuerpo humano conserva en el hígado. Las hormonas ayudan a mantener una constante concentración de glucosa en la sangre. Esto es especialmente importante para el cerebro porque no puede producir o almacenar glucosa, pero es dependiente de la glucosa suministrada por la sangre. Hasta los períodos breves de niveles bajos de glucosa (la hipoglucemia) puede causar daño cerebral. La insulina y el glucagons, son dos hormonas que secreta el pancreas, y regula los niveles de glucosa en sangre. La insulina desminuye la concentración de glucosa en la sangre; El glucagon lo aumenta. Dado a que la prevención de hipoglucemia es vital para el cuerpo humano, varias hormonas de las glándulas adrenales y la pituitaria respalda la function del glucagon. El consumo alcohólico interfiere con las tres fuentes de glucosa y con las acciones de las hormonas reguladoras. Los bebedores crónicos y empedernidos a menudo tienen un consumo dietético insuficiente de glucosa (8). Al no comer, las reservas de glicógeno se agotan en pocas horas (1). Además, la producción de glucosa corporal está inhibida, mientras el alcohol se está metabolizando (2). La combinación de estos efectos puede causar hipoglucemia severa de 6 a 36 horas después de un episodio (1) de un consumo del alcohol excesivo. Aun en personas bien nutridas, el alcohol puede modificar niveles de azúcar en sangre. El consumo alcohólico agudo, especialmente en combinación con azúcar, aumenta la secreción de insulina y causa hipoglucemia temporal (9). Además, los estudios en sujetos normales (10) y diabéticos dependientes a insulina (3) han demostrado que el consumo alcohólico agudo puede deteriorar la respuesta hormonal a la hipoglucemia. La ALERTA ALCOH�LICA. El instituto nacional en el Alcoholismo y Alcoholism No. 26 de octubre PH 352 1994.

El alcohol es una sustancia muy alta en calorías. Las grasas contienen 8 calorías por gramos, los cuáles son extraidos lentamente. Las proteínas y los carbohidratos (incluyendo azúcar blanco) contienen 4 calorías por gramo. El alcohol contiene 7 calorías por gramo y 20 % del alcohol es absorbido a través del endotelio del estómago. Para el hypoglycemico quién tiene bajos niveles de azúcar en sangre y quien experimenta síntomas incómodos, el alcohol es la forma más rápida para aumentar azúcar en la sangre. La hipoglucemia es facilmente identificada con una prueba de tolerancia de glucosa de cinco o seis horas. La hipoglucemia a menudo reacciona bien al cromo, el cinc, glutamina, y supplementación de magnesio y algunas veces a la supplementación de vitamina B3. Rogers y Pelton, (1957) encontró que la supplementación de glutamina redujo la ansiedad, el deseo para beber y mejoró el sueño, en una prueba de intercambio (crossover), doble ciego, de 6 semanas, con 7 hombres y 3 mujeres. Glutamina produjo mejorias en 9 de 10 bebedores que lo usaron. Durante la fase de intercambio del estudio, sólo 2 o 3 sujetos reaccionaron a la lactosa placebo. Glutamina es el aminoácido más abundante, en el cuerpo humano, y el cuerpo humano contiene aproximadamente 100,000 miligramos de glutamina. Glutamina probablemente ayuda al alcohólico en un minimo de 3 formas diferentes. Puede ser transformado en glucosa en sangre y la glucosa de sangre es el único combustible del cerebro, lo cual usa al menos 20 % de la energía del cuerpo humano y requiere un constante suministro de glucosa de sangre. A diferencia de grasas y carbohidratos, cuando la glutamina no es metabolizada para suministrar glucosa en sangre, la glutamina aumenta producción de proteínaa y la liberación de la hormona de crecimiento humana. La Glutamina también puede transformarse en el neurotransmisor exitativo glutamato o el neurotransmitter inhibitorio, acido gamma-aminobutyrico a merced del nivel exitatorio necesario. La Glutamina también acelera la reparación del endotelio intestinal, lo cual es afectado por alcohol.

Las Alergias Alimenticias

La adicción alcohólica también puede ser perpetuada por alergias ocultas a alimentos. Las alergias de comida a menudo producen deseos para las comidas que causan la alergia. William Philpott, Maryland, PhD certificado por la Junta Directiva de Neurología, Alergia y Psiquiatría, explica que el cuerpo humano produce endorphinas o enkephalinas, cuando es estresado por un alergeno. Las endorphinas parecidas a los opiodes, reduce ansiedad y perpetúa la adicción. Joseph Beasley, et al.(1991), examino 108 Alcohólicos para alergias en un programa de tratamiento en Amityville, Nueva York y encontro alergias en los siguientes porcentajes:Al maíz, 75.9 por ciento; a la leche, 74.1 por ciento; Al maní 67.6 el por ciento; Al cacao, 66.7 por ciento; Al huevo entero, 60.2 por ciento; A la naranja, 57.4 por ciento; Al el tomate, 52.8 por ciento; A el camarón, 48.1 por ciento; Y a el trigo 46.3 el por ciento. Beasley no examinó la alergia acia el centeno, pero otros investigadores han encontrado un porcentaje alto de alcohólicos alérgicos al centeno. Muchos bebidas alcoholicas son de maíz fermentados, trigo y centeno. La destilación puede concentrar a los cogeners que causa alergia, presentes en estos alimentos. Las alergias ocultas para estas comidas pueden perpetuar estas adicciones o pueden provocar comportamiento violento o imprevisible, lo cual es atípico para el bebedor.

Estas alergias a menudo se pueden detectar con una prueba de ELISA/ACT. Esta prueba puede detectar reacciones tardias de Immunoglobin G a 100 comidas diferentes, con una prueba de sangre. Dado a que la prueba ELISA/ACT es realizada en sangre, en lugar de piel, no hay que evitar el consumo de ninguna comida en prueba por un mínimo de cuatro días, mientras se realiza la prueba. La eliminación completa de un alergeno sospechoso de un alimento,durante un mínimo de cuatro días, permanece el método más preciso de probar una alergia encubierta de un alimento, y todavía puede ser necesario, si la experimentación preliminar indica una reacción alérgica. La prueba de eliminación de un alimento requiere evitar cualquier alimento en question de una forma diligente y estricta, hasta el conteniendo sospechóso de todos los ingredientes en sus etiquetas se debe repasar. Dado a que muchos de estos alergenos en alimentos son difíciles de evitar en los regímenes típicos del Americano. Las prubas de alergias ayuda a los pacientes darse cuenta de la necesidad de un cambio dietético. Las alergias de comida a menudo ocurren, cuándo el alcohol daña el endotelio intestinal. La permeabilidad intestinal aumentada (el síndrome de intestino permeable) puede ocurrir cuando el endotelio intestinal se irrita. Las proteínas indigestas pueden filtrarse a través del epitelio intestinal perforado, y pueden provocar una respuesta inmune cuando complejos del antígeno hacia anticuerpos se forman. Estas reacciones inmune pueden ocurrir, donde la sangre transporta los antígenos causando depresión, dolores de cabeza, deterioro de la concentración, déficit de atención, artritis y otras patologias, que son difíciles para asociarse con la comida ingerida.

La Depresión

La terapia antidepresiva comunmente ayuda y puede incluir supresores de reabsorbcion selectivo de la serotonin como Prozac, Zoloft, Effexor, Paxil, o Lexapro, o antidepresivos tricíclicos como Elavil y Pamelor, o supresores de monoamino oxidasa. Los suplementos nutritivos o herbarios, S-Adenosyl methionina S-Adenosyl, ampoya de San John (yerba), el aminoácido tryptophano, 5-hydroxytryptophano, tyrosina, y phenylalanine, también pueden ser utiles. Desafortunadamente, algunos fabricantes no han suministrado suplementos herbarios en la cantidad especificada en la etiqueta o en su publicidad. Los laboratorios del consumidor prueban los suplementos nutritivos y herbarios, de diversos fabricantes y publican los resultados, en Algunos resultados están disponibles gratuitamente para cualquiera, mientras que todos los resultados están disponibles, para el suscriptor.


Niveles alterados de tryptophano y serotonina ocurre en alcohólicos. La deficiencia del Neurotransmisor Seratonina, puede producir depresión. Tryptophano es un aminoácido esencial, lo cual quiere decir que el cuerpo humano no lo puede producir, aunque es necesario para producir otros aminoácidos. Tryptophano está disponible por receta, como L-Tryptophano o como el suplemento disponible sin receta, 5-Hydroxytryptophan. (5-HTP). El tryptophano es transformado a niacina (la Vitamina B3) o a el neurotransmitter serotonina en el cuerpo humano. Si un nivel inadecuado de niacina esta presente, tryptophano se convertira a niacina, antes de la conversión para serotonin. La conversión de tryptophano a serotonin requiere de Vitamin B6 (pyrodoxal 5 el fosfato) y magnesio, como cofactors. El alcohol agota magnesio y Vitamin B6. La investigación por Borg, S, et. al., (1985) indica que el alcohol aumenta niveles de serotonina durante la intoxificación, pero puede disminuir niveles de serotonina meses después de que el alcohólico vuelve sobriedad. Borg midió el producto metabólico serotonico, 5-Hydroxyindoleacetico (5-HIAA), en fluido cerebroespinal en sujetos controles y alcohólicos masculinos. 5-HIAA incremento con niveles de alcohol en sangre y gradualmente disminuido con la abstinencia. Los niveles subnormales ocurrieron en alcohólicos abstinentes durente tres meses.

Branchey L., et. al., (1985) observo niveles inferiores del tryptophano en plasma, en pacientes masculinos que tuvieron perdidad de conciencia, comparados a los pacientes que no experimentaron perdidad de conciencia. No hubo diferencia significativa entre las proporciones de otros aminoácidos que comparten el trasportador de tryptophano atraves de la barrera hematoencefalica. Aunque los niveles crecientes de la serotonina pueden beneficiar muchos alcohólicos en recuperacion, Bankole, A., et al., (2000) encontró que odansetrono, un antagonista 5-HT3 (que bloquea los receptores typo 3 de serotonina) disminuyo tragos al día, aproximadamente un 53 por ciento, en alcohólicos que comensaron a edad temprana.

Los niveles de Serotonina pueden ser aumentados con supplementación de l-tryptophano 5-hydroxytryptophan, además de fármacos.(P?Nger, W. et. al.,1991) comparo 5-hydroxytryptophano (5-HTP) a fluvoxamine (Luvox), un antidepresivo selectivo de reabsorbicion de serotonin, en un studio doble ciego de 6 semanas, con 69 participantes deprimidos. Ambos tratamientos produjeron reducciones altamente significativas en las Escaleas de Depresion de Hamilton, entre 2, 4 y 6 semanas. 5-HTP produjo mayores reducciones en el insomnio, en un 61.7 a 55.9 por ciento,en síntomas físicos un 47.6 a 37.8, en la ansiedad un 58.2 a 48.3, y redujo el estado de ánimo depresivo un 65.7 a 61.8, mas que con fluvoxamine. 5-HTP fue mejor tolerado, con cuatro pacientes recibiendo fluvoxamine que no termino el estudio, comparado con uno en el grupo 5-HTP, con un 38.9 por ciento de efectos secundarios precentes en el grupo de 5-HTP, comparado a un 54.5 por ciento del grupo de fluvoxamine quienes reportón efectos secundarios. Los efectos secundarios en el grupo 5-HTP fueron más leves y el que se retiro del grupo de 5-HTP, duró 5 semanas, mientras los 4 del grupo 4 fluvoxamine duraron 2 semanas. La dosis fueron de 100 mg 5-HTP tres veces al día o 50 mg de fluvoxamine 3 veces al día.

Amén, D y Routh, L. (2003) reporto que la reabsorbcion cerebral de 5-HTP es del 70 por ciento, mientras que la reabsorbcion cerebral del-tryptophan es sólo 3 por ciento. La información adicional referente al tratamiento de depresión con aminoácidos está disponible, en el sitio Web de Amen,

Phenylalanine es uno de los ocho aminoácidos esenciales y un precursor del fenilo de ethylamina (PEA). La deficiencia del PEA puede producir depresiones, que son insensibles al tryptophano, 5-HTP, tyrosina, SAME o a los antidepresivos de receta. Se ha encontrado niveles urinarios altos de PEA en schizophrenicos, mientras que han encontrados niveles bajos en pacientes deprimiós. La Phenylalanina también es un precursor de la tyrosina, y la tyrosina es un precursor para tironina (componente de la hormona tiroidea) y dopamine. Phenylalanine puede aumentar la presión sanguínea, o el pulso y no debe ser tomado si usted usa supresores de monoamino oxidase. (Beckmann, hidrógeno., et al., 1979) comporaron DL-PHENYLALANINA a imipramina, en un estudio doble ciego, de 40 pacientes deprimidos, de 30 días. Ninguna diferencia estadística fue encontrada, entre los dos tratamientos, en la Escala de Depresion de Hamilton y el cuestionario Bf-S de evalúacion propia. Las dosis usadas fueron de 150 a 200 miligramos por día de DL-PHENYLALANINE o imipramina.La Phenylalanina está disponible como Phenylalanina D, L o DL, pero más comúnmente esta disponible como DL-PHENYLALANINA. D-Phenylalanina inhibe a enkephalinasa, lo cual metaboliza enkephalinas y endorphinas y es usado en algunas fórmulas de propiedad para disminuir dolor y ansiedad. L-Phenylalanina es la composicion usada por el cuerpo humano para producir neurotransmisores y proteínas. DL Phenylalanine es una mezcla de Phenylalanina D y L.

La Tyrosine es un aminoácido de occurrencia natural y es un precursor para el neurotransmisor dopamina y thyronina (substrato para la hormona tiroidea). Daniel Amen, Maryland, es un psiquiatra que ha tomado sobre 28,000 tomografías del cerebro SPECT (escanografia). Amen reporta que la tyrosina ayuda contra la depresion, de dopamina reducida, y reduccion de flujo sanguíneo prefrontal de la corteza cerebral. Los niveles reducidos de dopamina ocurren en el déficit de atención, y las depression, donde el sueño, la fatiga, el letargo y el aumento del peso estan incrementados. Baja tensión arterial y ritmos cardíacos disminuidos pueden ocurrir por la deficiencia de dopamina. Tyrosina puede aumentar la presión sanguínea, o el pulso y no debería tomarse si usted usa supresores de monoamine oxidase.

Adenosyl metionina – S (SAMe) es producida naturalmente en el cuerpo humano, cruza la barrera hematoencefalica y ha resultado efectiva para tratar depresión y dolor articular. Tambien Aumenta niveles del serotonina y dopamina y mejora la fluidez de la membrana plasmática neuronal y cerebral. Ha aumentado episodios maníacos e hipo-maníacos, en pacientes con enfermedad bipolar y no debería ser usado por pacientes maníaco-depresivós. Bell, k, et al., (1994) condujo un estudio doble ciego, controlado por placebo, comparando SAMe a desipramina, en una prueba de 4 semanas con 26 pacientes deprimidos, en la Universidad de California, Irvine. SAMe mostro una mejoria en 62 por ciento de pacientes, comparado a un 50 por ciento de pacientes tratódos con desipramina. Sin tener en cuenta el tratamiento, los pacientes exhibiendo una disminución del 50 por ciento o más en La Escala de Hamilton, mostraron incrementos significativos de SAMe a niveles plasmaticos. Salmaggi, P, et. al., (1993) encontro una mejoria de mayor significancia en mujeres deprimidas post menopausicas tratadas con 1600 miligramos de la misma forma, que el placebo. Durante un prueba doble siega de 30 días, con 80 mujeres, entre 45 y 59 años de edad, se mostro una superioridad evidente en el día 10 y los efectos secundarios fueron leves. Amén, D. y Routh, L., (2003) tambien encontraron que SAMe son útil para la depresión. Jacob Teitelbaum, Maryland (2003) se recupero del síndrome crónico de fatiga, y recomienda de 200 a 800 miligramos de SAMe, dos veces al día, para pacientes con fatiga crónica depresiva y fibromyalgia, que muesta una mejoria en 10 días.

ST. Johns Wart
Saint Johns Wart es una hierba extensivamente investigada, prescrita por médicos en Europa para la depresión y la ansiedad. Ha resultado ser superior que el grupo placebo y al menos tan efectivo como amitriptylina, imipramina, maprotilina y paroxetina y usualmente produce menos efectos secundarios. El insomnio y la fotofobia, algunas veces, se aumentan con el uso de esta preparación herbaria. El efecto de St. Johns es similar a los supresores selectivos de reabsorbcion de serotonina, aunque dopamina, interleukina y los niveles de cortisol, también parecen ser afectados. Algunas formulas de St. Johns wart contienen Cadmio o la contaminación de plomo y otros no contienen las cantidades anunciadas o reclamadas de hypericum. A diferencia de tryptophano, 5-hydroxytryptophano, phenylalanina, tyrosina y de SAMe las cuáles naturalmente ocurren en el cuerpo humano, el Fermento de Malta de St. John interactúa con medicamentos. Zhou S, et. al. (2004) informa que el fermento de malta de St John disminuyó las concentraciones de amitriptylina, cyclosporina, digoxina, fexofenadina, indinavir, metadona, midazolam, nevirapina, phenprocoumon, simvastatina, tacrolimus, theophyllina y warfarina en sangre, mientras que no alteró la pharmacokinetica del ácido carbamazepina, dextromethorphano, mycophenolico y pravastatina. El fermento de malta de St John disminuyó la concentración de plasma del metabolito activo SN-38, en pacientes con cáncer recibiendo tratamiento con irinotecan. El fermento de malta de St John no alteró la pharmacokinetica de tolbutamida, pero aumentó la incidencia de hypoglycaemia. Varios casos han sido reportados que elFermento de Malta de St John disminuyó la concentración de cyclosporina en sangre conduciendo al rechazo de órganos. El fermento de malta de St John causó episodios de sangrando y embarazos imprevistos cuando se uso concomitantemente con píldoras anticonceptivas. También causó síndrome de serotonina cuando el co-administrado con supresores selectivos (por ejemplo el sertalina y paroxetina) de reabsorbcion de serotonina. Algunos alcohólicos y pacientes bipolares requieren tratamiento con un anticonvulsante, aun después de que los niveles adecuados de magnesio son restaurados. El ácido Valproico y gabapentina han resultado efectivos. En algunos casos la ansiedad persistirá. Acamprosate es poco adictivo y ofrece posibilidades nuevas para reducir la ansiedad del retiro alcohólico y la abstinencia anticipada. El insomnio es también común en el alcohólico abstinente. En algunos casos, los tranquilizantes pueden ser necesarios para ocuparse de insomnio, mientras melatonina, y supplementación de aminoácido son adecuados para otros.

Se ha estimado que Hypotiroidismo efecto hasta 10 por ciento de americanos de madia edad y puede producir depresión. Algunas víctimas del hypotiroidismo son incapaces de convertir la tetraiodotironina inactivo (T4), (levotironina, Synthroide) a la hormona tiroidea activa triiodotironina (T3), (liotironina, Cytomel). Muchos médicos asumen que los pacientes que no responden a tratamiento genérico de T4 no es hipo-tiroidea, sin intentar una prueba de Cytomel que es más cara. Frecuentemente, Cytomel mejorará el hypotiroidismo, cuándo el medicamento de T4 no lo hace. Cytomel actua rapidamente, con una vida media breve y los pacientes que no han mejorado con años de medicación T4, a menudo se sienten mejores el mismo día que consume Cytomel. La enfermedad bipolar que no reacciona a los demás medicamentos convencionales como el Litio o anticonvulsantes, algunas veces responden al medicamento T3. El medicamento litio puede causar hypotiroidism.

Las pruebas usuales del laboratorio para hormona tiroidea (TSH, T4, T3) algunas veces fracasan en detectar un hypotiroidismo, especialmente en la tiroiditis autoinmune de Hashimoto, lo cual viene aumentando progresivamente en su prevalecia. Las pruebas de fluorescencia más costosas de activacion de microespheras, algunas veces pueden detectar esta condición, donde el sistema inmunológico ataca tejido tiroideo.

Jacob Teitelbaum, (2005) informa que la Academia Americana de Endocrinólogos Clínicos redujo el rango normal para la hormona estimulante de tiroide (TSH), en noviembre, 2003. Un TSH de 3 o más, ahora es indicative de hypotiroidismo. Dado a que los criterios antiguos consideraban que el hypotiroidismo comenzar con un TSH de 5, en lugar de 3.0, el número de individuos hipo-tiroideos en los Estados Unidos, se estima haber aumentado de 13 al 26 millones. Teitelbaum indica menos de 25 por ciento de estas personas han sido verdaderamente diagnosticadas y tratadas. Teitelbaum discute el costo humano horrendo de hypotiroidism, en Comunicado de Prensa: La Epidemia Trágica e Invisible de Enfermedad Tiroidea.

La Testosterone
La mayor parte de alcohólicos son hombres y alcohól puede agotar la testosterona. Muchos varones entienen inmediatamente que éste podría producir depresión. Barrett-Connor, E., et. al., (1999) en un estudio de 856 hombres, 50 a 89 años de edad, se encontrao que el Inventario de Depresión de Beck difirió inversamente con niveles libres de la testosterona, independiente de edad, o actividad física. La testosterona Bioavailable fue 17 por ciento más baja, para los hombres deprimiós. Estadísticamente, la probabilidad de una diferencia tan grande, es de 7 en mil. (P = 0.007 el Alcohol agota el cinc gravamente y el cinc es necesario para la producción de la testosterona y la prevención de conversión de la testosterona a dihydrotestosterona, lo cual causa calvicie enlos hombres). El alcohol es directamente tóxico para los testiculos, que causa reducciones de la testosterona en hombres. En un estudio de hombres saludables normales que recibieron alcohol por 4 semanas, los niveles de la testosterona disminuyeron después de sólo 5 días y continuaron cayendo todo el período del estudio (17). La deficiencia prolongada de la testosterona puede contribuir a una “femininización” de las características sexuales propias de los hombres, por ejemplo la ginecomastia (18). El instituto Nacional Alerta al alcohól en el Alcoholismo No. 26 de octubre PH 352 1994 Sarkar, D., et. al. (1998) observo, que ” los hombres Alcohólicos muestran una asociación positiva entre la presencia de gynecomastia clínicamente ostensible y niveles circulantes elevados de prolactina. Estos pacientes también muestran una elevación de niveles de estrógeno en plasma, lo cual – se cree – es debido a la conversión periférica de andrógenos débiles a estrógenos “. La testosterona ha recibido mala prensa en los Estados Unidos. Gynecomastia o producción de tejido de ceno femenino en varones, es mencionado a menudo como un peligro de supplementación de la testosterona. La gynecomastia es fácilmente observable en alcohólicos crónicos mayors, mientras que es casi inexistentes en los gymnacios donde se alega que personas abusan esteroides. Los niveles aumentados de la testosterona parecen impedir hipertrofia beningna (BPH) de la próstata. BPH es una ampliación de la glándula prostática no maligna, lo cual interfiere con flujo de orina, en hombres mayores. Cuando la glándula prostática se amplia, constriñe la uretra. El sueño deteriorado ocurre, cuándo los hombres necesitan orinar frecuentemente durante la noche. BPH es asociado con elevacion de estrógeno y niveles disminuidos de testosterona. BPH ocurre en hombres mayors, o de edad media, cuando los niveles de estrógeno aumentan, mientras los niveles de la testosterona decrecen. En Francia, BPH es tratado con supplementación de testosterona.

La supplementación de la testosterona aparenta ser segura cuando es limitada a restaurar la testosterona a niveles fisiológicamente normales. La nueva testosterona transdérmica y los geles evitan el sistema gastrointestinal, y imitan los niveles naturales fluctuantes diarios de la hormona, más estrechamente, que con inyecciones semanales o mensuales.

Descarga De Responsabilidad Médica

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Siento pesar se me ha requerido tan bastante para escribir la presente de GRACIAS. En noviembre pasado mi hermano vino a mí y pidió mi ayuda, después de 35 años de ser esclavo de la bebida, él quiso despabilarse. Comprobé varios programas Detox en mi área, pero ninguno pareció estar bien para él. A través de los años él había probado varios programas pero ninguno de ellos se ocupó del problema de la raíz. Él siempre reincidió dentro de un mes. Luego vi algo en TV en 2am en la mañana acerca de su programa. Soy ha sido una enfermera pues 25 años y yo sabemos que mi hermano no estaría vivo ahora de no ser por usted el programa. Él bebía 2 cajas de cerveza un día /ventaja. Su régimen constó de tres o cuatro “mordiscos” de comida un día. Él fue piel y huesos. Él fue incapaz de ir de más que 3 horas sin una bebida antes de que él obtuvo las sacudidas. Supe que mi hermano no vería la Navidad. Después de encontrarse a con usted y su personal y escuchar lo que usted tuvo para decir acerca de adulto ADD y alcohol /drogas todo ello iniciados para hablar racionalmente. Por primera vez desde que mi hermano estaba en sus adolescentes alguien sentido hecho para él. Como usted puede acordarse de que mi hermano comenzó a beber a la edad de 18 después de la muerte de mi de 7 años de edad hermana a quien él era cuidar niños. Mi hermano menor salió a hurtadillas de la casa y fue golpeado por un coche. Siento como usted me devolvió a un hermano que perdí años atrás. GRACIAS con todo mi corazón. Él ha estado sobrio para 1 año ahora y va muy bien. Tardo cada oportunidad en contarle todo el mundo sobre su programa y su personal maravilloso. Mi hermano no ha extrañado uno dele seguimiento a la visita en un año, esto de un hombre que no había visto a un doctor en 15 años.

Simplemente sobre 7 meses que atrás estaba verdaderamente de cara a “mi �LTIMA LLAMADA por alcohol”. Tenía arritmias del corazón, pequeños temblores, sudores, vomitando diariamente, El laboratorio aprecia en un rango peligroso; Moría en el sentido literal y clínico. Quise comprender excepto didn’t, y fui demasiado agotado para ARREGLARSE mí mismo controlando y derrotando a este LIQUID. Estaba fuera de energía, espíritu y esperanza. Todo el mundo alrededor de mí estaba siendo torturado por esfuerzos fallidos, desamparo y confusión. Y luego fui guiado para Florida Detox® con Dr. Ä¡Sponaugle y su experto forman en equipo por mi Dave Debbie de hermana y del marido que dejó este milagro ocurrir!

…El avance rápido para ahora. Estoy tan contento para estar VIVITO Y COLEANDO. No desafilando o aletargando mis sentidos y apreciando mucho a cada uno y cada día. Ninguno de los deseos simplemente una voluntad a vivir bien y aprender de esa oscuridad divide de mi historia. Hay una razón para esto y el tiempo revelará lo que esa razón es; Tal vez a devolver para los otros en los que pueden estar o muy cerca para su personal última Llamada. No sé pero estoy tan contento para estar despierto y lo suficientemente vivo si que esa razón se aclarara soy capaz de saberla. Estoy tan contento para estar de regreso. La buena la de día y la malas así como la vida – se suponga – son. Es tal carga levantada no constantemente mentir. Esa culpabilidad es TAN pesada que es como ser aplastada. Usando mi cerebro, no quebrándose le promete, no escondiendo del mundo, todo justo una lista de la parte pequeña de vida diaria soy tan agradecido para.

Soy una persona que es una escéptica de algo que es demasiado buena para ser verdadera, Pero el MILAGRO que me ocurrió en Florida Detox® ha sido incluso mayor que mis expectativas más altas y sigue para mi familia también.

Dr Sponaugle y Pareja tuvieron los instintos para llegar al corazón de hay que comenzar a curarse. Mirando todo continuando a conmigo como una persona entera y con tal que lleve a cabo planes para estar entero para el futuro.

Agradeceré eternamente a quienes me ayudaron a llegar así de lejanos y esperar que en cierta forma pueda dar de regreso a todos y cada uno de usted algo así de realmente y sin precio como este regalo que usted me ha dado.

Ä¡Crea … es posible!
Fui justo a punto de comenzar a cocinar cena para mi familia y la mezcla mi primera bebida cuando enloquecí a través de los canales de la TV y aterricé en El Dr. Phil salga a la vista último noviembre. 2005. El tema estaba en madres que abusaron de drogas y alcohol. Fueron atractivos y condujeron sus minifurgonetas llenas de niños para instruir, lecciones de música y gastar las fiestas de noche. SINO, la mayoría de las veces estaban ‘ bajo la influencia ‘ de su adictivo particular de elección. Mi elección fue alcohol, escocesa para ser exacta. Mi corazón comenzó a palpitar más rápido y mi pulso aligerado como me di cuenta de que yo ya no pude negar mi adicción.

Durante el día, fui la madre capaz, alistándose como voluntario en 3 escuelas y grupos de estudio de la Biblia aun delanteros. Pero en la noche, racionalicé que brindé por “capa pluvial con criar a 3 adolescentes”. La hora de coctel se convirtió en “el Borracho de mamá otra vez” y mi marido me timonearía para el dormitorio y yo me desmayaría. Ä¿El gran ejemplo para colocar para mis niños, huh? Cada mañana tendría que preguntarme lo que le había dicho a quién y se llenaría de verg�enza. Me disculparía y prometería solemnemente para mí mismo para no hacerlo nuevamente hoy. Por años intenté detenerme o controlar mi vicio de la bebida. Le rogué a Dios que substraiga el sabor que había desarrollado para alcohol pero yo continué bebiendo. Hay un verso en la Biblia que dice, “como un perro regresa a Su vómito, así un tonto para su insensatez”. Oficialmente me había convertido en un tonto.

Llamé a Fl. Detox y hecho un appt. a entrar para el tratamiento adentro enero. 2006. Bebí todo directo los días de fiesta de Navidad, las celebraciones Nuevas de Año y hasta la noche antes de que me registrase para la clínica del hospital. Mi marido me trajo con mis bolsas como sardinas en lata y un cheque de caja. Me hice volver en sí a mí mismo con un corazón lleno de esperanza y un deseo profundo convertido en una persona de todo. Estaba aburrido de llevar puestas dos máscaras diametralmente opuestas. Mi vida fue una MENTIRA y mi autoestima se fue.

El personal me trató como si fuera un invitado honrado, no la escoria que me creí ser. Las enfermeras levantadas en el piso 7 trataron mi cuerpo humano con mansedumbre y me hablaron con compasión. Estaba desesperado continuamente por su preocupación genuina para mi comodidad y su respuesta rápida para mis preguntas o mis necesidades.

He sido alcohólico gratuitamente desde el día que dejé el hospital. Tengo la gran buenaventura para vivir acerca del paseo en coche de una hora de la clínica y esperar con anticipación mis visitas mensualmente de seguimiento. Cada día de mi sobriedad es un regalo precioso para mí y no deseo ardientemente alcohol en absoluto. Alguna vez no quiero ser esa mujer otra vez…….Estoy entero ahora. Llevar puestas máscaras y hacerse pasar por alguien que no fui fue justo también emotivamente agotando.

Dios le ha dado a Dr. Amontone en almiares el regalo de sabiduría médica para exactamente diagnosticar y en forma segura desintoxicar a los pacientes que quieren estar libres de la prisión de adicción de droga /alcohol. Dios ha puesto al personal en el lugar a expertamente llevar a cabo el programa de tratamiento de cada paciente durante su estadía. Pero sobre todo para mí, Dios tiene a Dr dotado. Rick con un corazón lleno de misericordia y lovingkindness hacia esos que Dios tiene, le confió a su cautela. Con este fin, seguramente recomendaría a Fl. Detox para alguien tratando de vivir una vida victoriosa gratuitamente de alcohol.

Gracias, Florida Detox®, para devolverme mi vida. El descanso de favor de mayo Dios en usted, le fortalece y protege a usted y su personal en todo lo al que usted pone su mano.

© Copyright (2012) Marvin Sponaugle, MD All Right Reserved